F. Geissmann

Immunology · 2000–2016

A researcher in the Science corpus: 23 papers; trusts 11 researchers and is trusted by 39 researchers here, as inferred from the citation graph.

Subfields

Whom F. Geissmann trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. N. Serbina
  2. S. Gordon
  3. Steffen Jung
  4. C. Auffray
  5. F. Tacke
  6. I. Mellman
  7. J. Matsuda
  8. I. Crispe

Who trusts F. Geissmann overall

  1. M. Guilliams
  2. S. Yona
  3. P. Italiani
  4. C. Shi
  5. N. Serbina
  6. S. Gordon
  7. C. Auffray
  8. C. Varol
  9. Eric G. Pamer
  10. C. Lumeng

Papers from F. Geissmann's perspective

Ordered by how strongly the researchers F. Geissmann trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Blood monocytes consist of two principal subsets with distinct migratory properties. (Immunity)
  2. Analysis of Fractalkine Receptor CX3CR1 Function by Targeted Deletion and Green Fluorescent Protein Reporter Gene Insertion (Molecular and Cellular Biology)
  3. A clonogenic common myeloid progenitor that gives rise to all myeloid lineages (Nature)
  4. Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2 (Nature Immunology)
  5. In vivo depletion of CD11c+ dendritic cells abrogates priming of CD8+ T cells by exogenous cell-associated antigens. (Immunity)
  6. Subpopulations of Mouse Blood Monocytes Differ in Maturation Stage and Inflammatory Response1 (Journal of Immunology)
  7. Fractalkine Preferentially Mediates Arrest and Migration of CD16+ Monocytes (Journal of Experimental Medicine)
  8. TNF/iNOS-producing dendritic cells mediate innate immune defense against bacterial infection. (Immunity)
  9. Gene expression profiles and transcriptional regulatory pathways underlying mouse tissue macrophage identity and diversity (Nature Immunology)
  10. Chemokines acting via CXCR2 and CXCR4 control the release of neutrophils from the bone marrow and their return following senescence. (Immunity)

Their papers