N. Serbina

Immunology · 2003–2008

A researcher in the Science corpus: 3 papers; trusts 7 researchers and is trusted by 19 researchers here, as inferred from the citation graph.

Subfields

Whom N. Serbina trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Colonna
  2. Steffen Jung
  3. F. Geissmann
  4. Eric G. Pamer
  5. G. Trinchieri
  6. I. Charo
  7. J. Galán
  8. F. Fang
  9. F. Tacke
  10. S. Luther

Who trusts N. Serbina overall

  1. C. Auffray
  2. Eric G. Pamer
  3. L. Chávez-Galán
  4. P. Murray
  5. M. Lowes
  6. C. Shi
  7. A. Mildner
  8. F. Geissmann
  9. P. Italiani
  10. M. Guilliams

Papers from N. Serbina's perspective

Ordered by how strongly the researchers N. Serbina trusts endorse each paper. Same corpus, a different order for every researcher.

  1. In vivo depletion of CD11c+ dendritic cells abrogates priming of CD8+ T cells by exogenous cell-associated antigens. (Immunity)
  2. Analysis of Fractalkine Receptor CX3CR1 Function by Targeted Deletion and Green Fluorescent Protein Reporter Gene Insertion (Molecular and Cellular Biology)
  3. Blood monocytes consist of two principal subsets with distinct migratory properties. (Immunity)
  4. Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2 (Nature Immunology)
  5. A Coordinated Change in Chemokine Responsiveness Guides Plasma Cell Movements (Journal of Experimental Medicine)
  6. TNF/iNOS-producing dendritic cells mediate innate immune defense against bacterial infection. (Immunity)
  7. Intrasplenic steady-state dendritic cell precursors that are distinct from monocytes (Nature Immunology)
  8. Distinct dendritic cell populations sequentially present antigen to CD4 T cells and stimulate different aspects of cell-mediated immunity. (Immunity)
  9. Subpopulations of Mouse Blood Monocytes Differ in Maturation Stage and Inflammatory Response1 (Journal of Immunology)
  10. A clonogenic common myeloid progenitor that gives rise to all myeloid lineages (Nature)

Their papers