Correction: Corrigendum: Constant replenishment from circulating monocytes maintains the macrophage pool in the intestine of adult mice
By C. Bain, A. Bravo-Blas, Charlotte L. Scott, Elisa Gomez Perdiguero, F. Geissmann, S. Henri, B. Malissen, L. Osborne, D. Artis, A. Mowat. Nature Immunology, 2014
205 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/6fa6a7abbf51a908fdcc924501278d6d9e019649
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Citations in the corpus
- Homeostatic immunity and the microbiota (2017, unknown): “The microbiota also control the homeostatic replenishment of monocytederived macrophages in the intestinal mucosa (Bain et al., 2014).”
- Macrophage Polarization. (2017, background): “In the gut, macrophage turnover is rapid (days to weeks), whereas in the lungs, months to years are needed to replace the embryonic-derived alveolar macrophages (51, 53, 55, 56).”
- Macrophages: development and tissue specialization. (2015, background): “Importantly, it has recently been shown that yolk sac– and fetal liver–derived resident intestinal lamina propria macrophages are present during the neonatal period, but they fail to persist in the adult intestine and are completely replaced at the time of weaning by BM-derived Ly6Chi monocytes (109”
- Colony-stimulating factor 1 receptor (CSF1R) inhibitors in cancer therapy (2017, background): “Intestinal macrophages also originate from monocytes [51] but exhibit a reduced inflammatory activation state that allows the healthy co-existence with commensal bacteria achieved by locally produced autocrine IL-10 as well as regulatory T-cell-derived IL-10 production [52].”
- The fate and lifespan of human monocyte subsets in steady state and systemic inflammation (2017, background): “Classical monocytes replenish the large resident monocyte-derived population of the gut (Bain et al., 2014) and skin (Tamoutounour et al., 2013) and are poised to migrate to sites of inflammation, where they display D ow nloaded from https://rupress.org/jem /article-pdf/214/7/1913/545218/jem…”
- The interaction of anticancer therapies with tumor-associated macrophages (2015, background): “1), with the exception of the adult hematopoietic origin of gut, heart, and dermis macrophages (Bain et al., 2014; McGovern et al., 2014; Molawi et al., 2014).”
- Gut microbiota: Role in pathogen colonization, immune responses, and inflammatory disease (2017, background): “All rights reserved around the time of weaning, in a process that is dependent on CCR2 expression of the monocytes and neonatal gut microbiota (79).”