D. Cua

Immunology · 2002–2019

A researcher in the Science corpus: 28 papers; trusts 32 researchers and is trusted by 45 researchers here, as inferred from the citation graph.

Subfields

Whom D. Cua trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. E. Bettelli
  2. S. Gaffen
  3. M. McGeachy
  4. M. Veldhoen
  5. T. Korn
  6. R. Mebius
  7. L. Harrington
  8. J. Kolls
  9. I. Ivanov
  10. Yi Chen

Who trusts D. Cua overall

  1. Ming O. Li
  2. S. Gaffen
  3. Wenjun Ouyang
  4. M. McGeachy
  5. C. Tato
  6. J. Kolls
  7. P. Miossec
  8. M. Sospedra
  9. Y. Iwakura
  10. R. Flavell

Papers from D. Cua's perspective

Ordered by how strongly the researchers D. Cua trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
  2. Microbial Lipopeptides Induce the Production of IL-17 in Th Cells1 (Journal of Immunology)
  3. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  4. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
  5. Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells (Nature Immunology)
  6. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
  7. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
  8. A crucial role for interleukin (IL)-1 in the induction of IL-17–producing T cells that mediate autoimmune encephalomyelitis (Journal of Experimental Medicine)
  9. A human interleukin-12/23 monoclonal antibody for the treatment of psoriasis. (New England Journal of Medicine)
  10. A novel transcription factor, T-bet, directs Th1 lineage commitment. (Cell)

Their papers