M. McGeachy

Immunology · 2002–2019

A researcher in the Science corpus: 8 papers; trusts 6 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom M. McGeachy trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. E. Bettelli
  2. Shimon Sakaguchi
  3. Benjamin N. Gantner
  4. D. Cua
  5. E. Shevach
  6. Xuexian O. Yang
  7. L. Harrington
  8. Yi Chen
  9. I. Ivanov
  10. S. Gaffen

Who trusts M. McGeachy overall

  1. Ming O. Li
  2. R. Flavell
  3. D. Cua
  4. Y. Lee
  5. Liang Zhou
  6. S. Gaffen
  7. E. Shevach
  8. Jinfang Zhu
  9. J. Goverman
  10. A. Chaudhry

Papers from M. McGeachy's perspective

Ordered by how strongly the researchers M. McGeachy trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12.
  2. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
  3. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  4. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
  5. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
  6. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
  7. Microbial Lipopeptides Induce the Production of IL-17 in Th Cells1 (Journal of Immunology)
  8. Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
  9. A human interleukin-12/23 monoclonal antibody for the treatment of psoriasis. (New England Journal of Medicine)
  10. Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells (Nature Immunology)

Their papers