I. Ivanov

Molecular Biology · 2006–2015

A researcher in the Science corpus: 8 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

Whom I. Ivanov trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. S. Mazmanian
  2. R. Sartor
  3. A. Macpherson
  4. C. Weaver
  5. Paul R. Mangan
  6. Ruth E. Ley
  7. Brigitta Stockinger
  8. Yan Zheng
  9. L. Harrington
  10. M. Veldhoen

Who trusts I. Ivanov overall

  1. K. Honda
  2. Dan R. Littman
  3. Y. Belkaid
  4. Hsin-jung Wu
  5. G. Lee
  6. Y. Lee
  7. June L. Round
  8. Jinfang Zhu
  9. S. Mazmanian
  10. K. Matsuoka

Papers from I. Ivanov's perspective

Ordered by how strongly the researchers I. Ivanov trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
  2. Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens (Nature Medicine)
  3. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
  4. An immunomodulatory molecule of symbiotic bacteria directs maturation of the host immune system. (Cell)
  5. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  6. Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)
  7. Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells. (Proceedings of the National Academy of Sciences of the United States of America)
  8. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
  9. An essential function for the nuclear receptor RORγt in the generation of fetal lymphoid tissue inducer cells (Nature Immunology)
  10. Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)

Their papers