C. Tato

Immunology · 2006–2020

A researcher in the Science corpus: 12 papers; trusts 5 researchers and is trusted by 6 researchers here, as inferred from the citation graph.

Subfields

• Immunology — 185 citations into it; #660 of 2,160 by trust here, #194 by citations
• Pathology and Forensic Medicine — 53 citations into it; #243 of 414 by trust here, #182 by citations
• Cancer Research — 5 citations into it

Whom C. Tato trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Cua
2. M. Veldhoen
3. A. Laurence
4. R. Mebius

Who trusts C. Tato overall

1. M. McGeachy
2. D. Cua
3. A. Jetten
4. T. Malek
5. Jinfang Zhu
6. Liang Zhou
7. T. Hirano

Papers from C. Tato's perspective

Ordered by how strongly the researchers C. Tato trusts endorse each paper. Same corpus, a different order for every researcher.

1. Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
2. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
3. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
4. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
5. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
6. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
7. Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells (Nature Immunology)
8. Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
9. Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells. (Proceedings of the National Academy of Sciences of the United States of America)
10. Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12.

Their papers

• Single-cell transcriptomics of 20 mouse organs creates a Tabula Muris (2018) — 2,436 citations
• A Single Cell Transcriptomic Atlas Characterizes Aging Tissues in the Mouse (2020) — 984 citations
• IDseq—An open source cloud-based pipeline and analysis service for metagenomic pathogen detection and monitoring (2020) — 329 citations
• IL-23-Independent IL-17 Production Regulates Intestinal Epithelial Permeability (2015) — 712 citations
• Generation of Pathogenic Th17 Cells in the Absence of TGF-β Signaling (2010) — 1,234 citations
• Innate IL-17-producing cells: the sentinels of the immune system (2010) — 776 citations
• Innate IL-17-producing cells: the sentinels of the immune system (2010) — 1,018 citations
• Interleukin 23 receptor is essential for terminal differentiation of effector T helper type 17 cells in vivo (2009) — 1,081 citations
• Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation. (2007) — 1,600 citations
• TGF-β and IL-6 drive the production of IL-17 and IL-10 by T cells and restrain TH-17 cell–mediated pathology (2007) — 1,513 citations
• Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (2006) — 1,042 citations
• Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells. (2006) — 712 citations