Wenjun Ouyang

Immunology · 2000–2020

A researcher in the Science corpus: 15 papers; trusts 9 researchers and is trusted by 7 researchers here, as inferred from the citation graph.

Subfields

Whom Wenjun Ouyang trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Lowes
  2. D. Cua
  3. Yi Chen
  4. A. O’Garra
  5. Yan Zheng
  6. W. Strober
  7. Y. Belkaid
  8. W. Blumenschein
  9. E. Shevach
  10. M. Veldhoen

Who trusts Wenjun Ouyang overall

  1. Nikolaos G. Frangogiannis
  2. S. Bromley
  3. Jinfang Zhu
  4. I. Ivanov
  5. H. Spits
  6. A. Chaudhry
  7. H. Ishigame
  8. T. Mogensen
  9. Kole T. Roybal
  10. T. Mempel

Papers from Wenjun Ouyang's perspective

Ordered by how strongly the researchers Wenjun Ouyang trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
  2. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  3. Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
  4. Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain (Nature)
  5. IL-22 increases the innate immunity of tissues. (Immunity)
  6. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
  7. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
  8. Treatment with a neutralizing anti-murine interleukin-17 antibody after the onset of collagen-induced arthritis reduces joint inflammation, cartilage destruction, and bone erosion. (Arthritis & Rheumatism)
  9. Blockade of interleukin 6 trans signaling suppresses T-cell resistance against apoptosis in chronic intestinal inflammation: Evidence in Crohn disease and experimental colitis in vivo (Nature Medicine)
  10. Activated macrophages are an adaptive element of the colonic epithelial progenitor niche necessary for regenerative responses to injury. (Proceedings of the National Academy of Sciences of the United States of America)

Their papers