G. Clarke

Molecular Biology · Molecular Psychiatry · 2008–2022

A researcher in the Science corpus: 18 papers; trusts 17 researchers and is trusted by 12 researchers here, as inferred from the citation graph.

Subfields

Whom G. Clarke trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. J. Cryan
  2. Ruth E. Ley
  3. Ted G. Dinan
  4. Peter J. Turnbaugh
  5. F. Bäckhed
  6. Emeran A. Mayer
  7. Jeffrey I. Gordon
  8. S. Grenham
  9. S. O'Mahony
  10. Elizabeth A. Archie

Who trusts G. Clarke overall

  1. Ted G. Dinan
  2. J. Cryan
  3. P. Kennedy
  4. R. Stilling
  5. T. Sampson
  6. Yan Wang
  7. J. Foster
  8. J. R. Kelly
  9. S. Mazmanian
  10. Ygor P. Silva

Papers from G. Clarke's perspective

Ordered by how strongly the researchers G. Clarke trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Ecological and evolutionary forces shaping microbial diversity in the human intestine. (Cell)
  2. Obesity alters gut microbial ecology. (Proceedings of the National Academy of Sciences of the United States of America)
  3. The gut microbiota as an environmental factor that regulates fat storage. (Proceedings of the National Academy of Sciences of the United States of America)
  4. Development of the Human Infant Intestinal Microbiota (PLoS Biology)
  5. Molecular-phylogenetic characterization of microbial community imbalances in human inflammatory bowel diseases (Proceedings of the National Academy of Sciences of the United States of America)
  6. Worlds within worlds: evolution of the vertebrate gut microbiota (Nature Reviews Microbiology)
  7. Microbial ecology: Human gut microbes associated with obesity (Nature)
  8. Normal gut microbiota modulates brain development and behavior (Proceedings of the National Academy of Sciences of the United States of America)
  9. Comparative Metagenomics Revealed Commonly Enriched Gene Sets in Human Gut Microbiomes (DNA Research)
  10. Host-mediated inflammation disrupts the intestinal microbiota and promotes the overgrowth of Enterobacteriaceae. (Cell Host and Microbe)

Their papers