V. Dixit
Molecular Biology · 2000–2020
A researcher in the Science corpus: 23 papers; trusts 27 researchers and is trusted by 28 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 505 citations into it; #1,727 of 17,773 by trust here, #351 by citations
- Immunology — 298 citations into it; #455 of 2,160 by trust here, #163 by citations
- Epidemiology — 115 citations into it
- Cancer Research — 94 citations into it; #1,761 of 1,780 by trust here, #354 by citations
Whom V. Dixit trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- C. A. Dinarello
- Edward A. Miao
- Fabio Martinon
- J. Tschopp
- F. Bauernfeind
- O. Takeuchi
- V. Hornung
- S. Mariathasan
- Rongbin Zhou
- S. Riedl
Who trusts V. Dixit overall
Papers from V. Dixit's perspective
Ordered by how strongly the researchers V. Dixit trusts endorse each paper. Same corpus, a different order for every researcher.
- Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf (Nature)
- Gout-associated uric acid crystals activate the NALP3 inflammasome (Nature)
- Silica crystals and aluminum salts mediate NALP-3 inflammasome activation via phagosomal destabilization (Nature Immunology)
- Immunological and inflammatory functions of the interleukin-1 family. (Annual Review of Immunology)
- Reconstituted NALP1 inflammasome reveals two-step mechanism of caspase-1 activation. (Molecules and Cells)
- Critical Role for Cryopyrin/Nalp3 in Activation of Caspase-1 in Response to Viral Infection and Double-stranded RNA*> (Journal of Biological Chemistry)
- Pyroptosis: host cell death and inflammation (Nature Reviews Microbiology)
- Nalp1b controls mouse macrophage susceptibility to anthrax lethal toxin (Nature Genetics)
- Thioredoxin-interacting protein links oxidative stress to inflammasome activation (Nature Immunology)
- The Nalp3 inflammasome is essential for the development of silicosis (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Cryopyrin activates the inflammasome in response to toxins and ATP (2006) — 3,164 citations
- Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf (2004) — 1,905 citations
- Non-canonical inflammasome activation targets caspase-11 (2011) — 2,444 citations
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 (2018) — 5,732 citations
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015 (2014) — 970 citations
- Noncanonical Inflammasome Activation by Intracellular LPS Independent of TLR4 (2013) — 1,456 citations
- De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-κB signalling (2004) — 1,832 citations
- Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling (2015) — 3,299 citations
- Apoptosis signaling. (2000) — 1,146 citations
- Inflammasomes and their roles in health and disease. (2012) — 910 citations
- Glyburide inhibits the Cryopyrin/Nalp3 inflammasome (2009) — 816 citations
- Mechanisms and functions of inflammasomes. (2014) — 2,315 citations
- IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis. (2007) — 949 citations
- Signaling in innate immunity and inflammation. (2012) — 1,518 citations
- GsdmD p30 elicited by caspase-11 during pyroptosis forms pores in membranes (2016) — 816 citations
- NINJ1 mediates plasma membrane rupture during lytic cell death (2020) — 675 citations
- Sensitivity to antitubulin chemotherapeutics is regulated by MCL1 and FBW7 (2011) — 781 citations
- Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival (2010) — 636 citations
- Mitochondrial reactive oxygen species drive proinflammatory cytokine production (2011) — 742 citations
- Activity of Protein Kinase RIPK3 Determines Whether Cells Die by Necroptosis or Apoptosis (2014) — 665 citations