Signaling in innate immunity and inflammation.
By K. Newton, V. Dixit. Cold Spring Harbor Perspectives in Biology, 2012
1,518 citations recorded by the source corpus.
Subfields: Immunology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/3519969eef4c19d2b80de87e6c7a618ccbd48440
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Immunology
- Sumanth D. Prabhu
- Ting Liu
- Lingyun Zhang
- Douglas R. Green
- Donghyun Joo
- Shao-Cong Sun
- F. Llambi
- Nikolaos G. Frangogiannis
In Molecular Biology
- F. Llambi
- Sumanth D. Prabhu
- Douglas R. Green
- Nikolaos G. Frangogiannis
- E. Pereira
- Todd D. Gould
- Ting Liu
- Lingyun Zhang
Citations in the corpus
- Blood-Contacting Biomaterials: In Vitro Evaluation of the Hemocompatibility (2018, background): “Thereby, the following conformational changes on the bloodcontacting surface are considered as initial trigger for the complement activation via the alternative or classical pathway (Gorbet and Sefton, 2004; Andersson et al., 2005; Nilsson et al., 2007).”
- Cell Death Signaling. (2015, background): “Through a series of homotypic interactions, these initiate theassemblyof largemacromolecularcomplexesthatrecruit and activate caspase-8 for apoptotic signaling, as well as other signaling molecules that control processes such as inflammation and cell adhesion (Newton and Dixit 2012).”
- Platelet-Rich Plasma: New Performance Understandings and Therapeutic Considerations in 2020 (2020, background): “There are many classes of PRRs, including Toll-like receptors (TLRs) and RIG-1-like receptors (RLRs) [108].”
- NF-κB signaling in inflammation (2017, positive): “18,20,21 The different families of PRRs have distinct structural properties and respond to different PAMPs and DAMPs, but they share many similarities in the downstream signal transduction pathways.”
- Ketamine and Ketamine Metabolite Pharmacology: Insights into Therapeutic Mechanisms (2018, background): “Release of proinflammatory cytokines by these cells then activate members of the adaptive immune system to initiate an inflammatory response (Newton and Dixit, 2012).”
- The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. (2016, background): “stranded RNA, single-stranded RNA, and complement, among others.(7,8,11,12,14) The PRRs are primarily the membrane-bound”