Claudio Franceschi
Molecular Biology · 2000–2020
A researcher in the Science corpus: 12 papers; trusts 3 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 135 citations into it; #8,718 of 17,773 by trust here, #4,843 by citations
- Physiology — 89 citations into it; #795 of 2,276 by trust here, #905 by citations
- Immunology — 48 citations into it
- Epidemiology — 39 citations into it; #797 of 2,148 by trust here, #737 by citations
Whom Claudio Franceschi trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- J. Campisi
- Steve Horvath
- T. Tchkonia
- G. Hotamışlıgil
- Jean-Philippe Coppé
- F. Rodier
- Margaret F. Gregor
- Adam Freund
- Yi Zhu
- J. Tidball
Who trusts Claudio Franceschi overall
- Siddhartha Jaiswal
- Paolo Garagnani
- Benjamin L. Ebert
- J. Campisi
- Luigi Ferrucci
- Anna Katharina Simon
- E. Montecino-Rodriguez
- I.Maeve Rea
- Jesús Gil
- Nicolás Herranz
Papers from Claudio Franceschi's perspective
Ordered by how strongly the researchers Claudio Franceschi trusts endorse each paper. Same corpus, a different order for every researcher.
- Inflammaging and anti-inflammaging: a systemic perspective on aging and longevity emerged from studies in humans. (Mechanisms of Ageing and Development)
- Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
- Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders (Nature)
- Gut microbiota composition correlates with diet and health in the elderly (Nature)
- The Senescence-Associated Secretory Phenotype: The Dark Side of Tumor Suppression (Annual Review of Pathology)
- Cellular senescence and the senescent secretory phenotype: therapeutic opportunities. (Journal of Clinical Investigation)
- A senescent cell bystander effect: senescence-induced senescence (Aging Cell)
- Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (PLoS Biology)
- Frailty in older adults: evidence for a phenotype. (The journals of gerontology. Series A, Biological sciences and medical sciences)
- Hallmarks of cancer: the next generation. (Cell)
Their papers
- Chronic inflammation (inflammaging) and its potential contribution to age-associated diseases. (2014) — 3,497 citations
- Through Ageing, and Beyond: Gut Microbiota and Inflammatory Status in Seniors and Centenarians (2010) — 1,401 citations
- Gut Microbiota and Extreme Longevity. (2016) — 868 citations
- Undulating changes in human plasma proteome profiles across the lifespan (2019) — 815 citations
- Mediterranean diet intervention alters the gut microbiome in older people reducing frailty and improving health status: the NU-AGE 1-year dietary intervention across five European countries (2020) — 810 citations
- Chronic inflammation in the etiology of disease across the life span (2019) — 3,886 citations
- Immunosenescence and Inflamm-Aging As Two Sides of the Same Coin: Friends or Foes? (2018) — 1,190 citations
- Inflammaging: a new immune–metabolic viewpoint for age-related diseases (2018) — 2,795 citations
- The Continuum of Aging and Age-Related Diseases: Common Mechanisms but Different Rates (2018) — 782 citations
- Inflammaging and 'Garb-aging'. (2017) — 842 citations
- Inflammaging and anti-inflammaging: a systemic perspective on aging and longevity emerged from studies in humans. (2007) — 2,034 citations
- Inflamm‐aging: An Evolutionary Perspective on Immunosenescence (2000) — 4,917 citations