Nicolás Herranz
Physiology · 2015–2018
A researcher in the Science corpus: 2 papers; trusts 7 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 71 citations into it
- Molecular Biology — 63 citations into it
- Oncology — 14 citations into it
- Dermatology — 6 citations into it
Whom Nicolás Herranz trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Joon-Il Jun
- T. Kuilman
- J. Campisi
- M. Demaria
- Reut Yosef
- Adam Freund
- Rémi-Martin Laberge
- L. Lau
- S. Yoshimoto
- D. Baker
Who trusts Nicolás Herranz overall
Papers from Nicolás Herranz's perspective
Ordered by how strongly the researchers Nicolás Herranz trusts endorse each paper. Same corpus, a different order for every researcher.
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Senescence-messaging secretome: SMS-ing cellular stress (Nature Reviews. Cancer)
- Senescence surveillance of pre-malignant hepatocytes limits liver cancer development (Nature)
- A complex secretory program orchestrated by the inflammasome controls paracrine senescence (Nature Cell Biology)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders (Nature)
- p38MAPK is a novel DNA damage response‐independent regulator of the senescence‐associated secretory phenotype (EMBO Journal)
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
- Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
Their papers
- mTOR regulates MAPKAPK2 translation to control the senescence-associated secretory phenotype (2015) — 708 citations
- Mechanisms and functions of cellular senescence. (2018) — 988 citations