Adam Freund

Molecular Biology · 2009–2015

A researcher in the Science corpus: 6 papers; trusts 15 researchers and is trusted by 6 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 107 citations into it; #5,453 of 17,773 by trust here, #2,948 by citations
• Physiology — 100 citations into it; #427 of 2,276 by trust here, #396 by citations
• Immunology — 42 citations into it; #1,789 of 2,160 by trust here, #1,196 by citations
• Oncology — 34 citations into it; #992 of 1,698 by trust here, #1,046 by citations
• Cancer Research — 24 citations into it; #1,360 of 1,780 by trust here, #758 by citations
• Aging — 18 citations into it
• Genetics — 12 citations into it

Whom Adam Freund trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Jean-Philippe Coppé
2. J. Campisi
3. F. Rodier
4. A. Krtolica
5. S. Parrinello
6. C. Patil
7. P. Desprez
8. V. Krizhanovsky
9. T. Kuilman
10. F. d’Adda di Fagagna

Who trusts Adam Freund overall

1. J. Campisi
2. M. Demaria
3. S. Glück
4. Alejandra Hernandez-Segura
5. Nicolás Herranz
6. Jan M. van Deursen
7. T. Tchkonia
8. Jesús Gil
9. R. Kumari
10. J. Nehme

Papers from Adam Freund's perspective

Ordered by how strongly the researchers Adam Freund trusts endorse each paper. Same corpus, a different order for every researcher.

1. Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
2. Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
3. Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
4. Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
5. Cellular Senescence in Aging Primates (Science)
6. Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints (Nature)
7. Senescence of activated stellate cells limits liver fibrosis (Cell)
8. Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (Nature Cell Biology)
9. Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
10. Accumulation of senescent cells in mitotic tissue of aging primates. (Mechanisms of Ageing and Development)

Their papers

• Lamin B1 loss is a senescence-associated biomarker (2012) — 933 citations
• Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (2009) — 2,520 citations
• Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype (2015) — 1,109 citations
• p38MAPK is a novel DNA damage response‐independent regulator of the senescence‐associated secretory phenotype (2011) — 983 citations
• MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation (2015) — 1,061 citations
• Inflammatory Networks during Cellular Senescence: Causes and Consequences (2010) — 1,311 citations