Jesús Gil
Molecular Biology · 2005–2020
A researcher in the Science corpus: 11 papers; trusts 38 researchers and is trusted by 6 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 308 citations into it; #13,753 of 17,773 by trust here, #2,137 by citations
- Physiology — 207 citations into it; #639 of 2,276 by trust here, #104 by citations
- Oncology — 73 citations into it; #1,539 of 1,698 by trust here, #1,080 by citations
- Immunology — 65 citations into it; #1,802 of 2,160 by trust here, #443 by citations
- Cancer Research — 55 citations into it
- Genetics — 32 citations into it
- Aging — 30 citations into it
- Epidemiology — 30 citations into it
Whom Jesús Gil trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- J. Campisi
- Jean-Philippe Coppé
- M. Demaria
- D. Baker
- N. Sharpless
- T. Kuilman
- Joon-Il Jun
- D. Muñoz‐Espín
- Rémi-Martin Laberge
- Bennett G. Childs
Who trusts Jesús Gil overall
Papers from Jesús Gil's perspective
Ordered by how strongly the researchers Jesús Gil trusts endorse each paper. Same corpus, a different order for every researcher.
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
- Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (PLoS Biology)
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
- Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
- Senescence-messaging secretome: SMS-ing cellular stress (Nature Reviews. Cancer)
- Senescence surveillance of pre-malignant hepatocytes limits liver cancer development (Nature)
- Tumour biology: Senescence in premalignant tumours (Nature)
Their papers
- Molecular Interplay of the Non-coding RNA ANRIL and Methylated Histone H3 Lysine 27 by Polycomb CBX7 in Transcriptional Silencing of INK4a (2010) — 1,435 citations
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (2008) — 1,732 citations
- Tumour biology: Senescence in premalignant tumours (2005) — 1,538 citations
- Senescence impairs successful reprogramming to pluripotent stem cells. (2009) — 648 citations
- mTOR regulates MAPKAPK2 translation to control the senescence-associated secretory phenotype (2015) — 708 citations
- Senescence and the SASP: many therapeutic avenues (2020) — 993 citations
- Mechanisms and functions of cellular senescence. (2018) — 988 citations
- Senescence and aging: Causes, consequences, and therapeutic avenues (2018) — 1,060 citations
- A complex secretory program orchestrated by the inflammasome controls paracrine senescence (2013) — 2,043 citations
- Senescence surveillance of pre-malignant hepatocytes limits liver cancer development (2011) — 1,493 citations
- Glycolytic enzymes can modulate cellular life span. (2005) — 661 citations