Jean-Philippe Coppé

Molecular Biology · 2004–2010

A researcher in the Science corpus: 6 papers; trusts 6 researchers and is trusted by 11 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 97 citations into it; #7,796 of 17,773 by trust here, #3,044 by citations
• Physiology — 77 citations into it; #228 of 2,276 by trust here, #219 by citations
• Aging — 20 citations into it

Whom Jean-Philippe Coppé trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. J. Campisi
2. A. Krtolica
3. S. Parrinello
4. Toren Finkel
5. F. d’Adda di Fagagna
6. T. Kuilman
7. R. Kortlever
8. I. Ben-Porath
9. U. Herbig
10. M. Collado

Who trusts Jean-Philippe Coppé overall

1. J. Campisi
2. Adam Freund
3. M. Demaria
4. F. Rodier
5. Alejandra Hernandez-Segura
6. Hui Yang
7. Bennett G. Childs
8. Tuo Li
9. C. Patil
10. Domhnall McHugh

Papers from Jean-Philippe Coppé's perspective

Ordered by how strongly the researchers Jean-Philippe Coppé trusts endorse each paper. Same corpus, a different order for every researcher.

1. Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
2. Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
3. Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
4. Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
5. Putting tumours in context (Nature Reviews. Cancer)
6. Tumour biology: Senescence in premalignant tumours (Nature)
7. Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
8. Oncogenic BRAF induces senescence and apoptosis through pathways mediated by the secreted protein IGFBP7. (Cell)
9. Reversal of human cellular senescence: roles of the p53 and p16 pathways (EMBO Journal)
10. Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)

Their papers

• Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (2008) — 3,837 citations
• Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (2009) — 2,520 citations
• The Senescence-Associated Secretory Phenotype: The Dark Side of Tumor Suppression (2010) — 4,730 citations
• Senescent cells as a source of inflammatory factors for tumor progression (2010) — 624 citations
• Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (2009) — 632 citations
• Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (2004) — 622 citations