Michael N. Hall
Molecular Biology · 2000–2018
A researcher in the Science corpus: 14 papers; trusts 52 researchers and is trusted by 46 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 313 citations into it; #788 of 17,773 by trust here, #908 by citations
- Physiology — 58 citations into it
- Cell Biology — 55 citations into it
- Cancer Research — 33 citations into it
- Epidemiology — 30 citations into it
- Immunology — 23 citations into it
- Genetics — 16 citations into it; #1,826 of 6,854 by trust here, #976 by citations
- Biochemistry — 14 citations into it
Whom Michael N. Hall trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- K. Inoki
- R. Loewith
- Y. Sancak
- S. Wullschleger
- Christian C. Dibble
- Dos D. Sarbassov
- Roberto Zoncu
- L. Bar-Peled
- E. Jacinto
- David M. Sabatini
Who trusts Michael N. Hall overall
- Asier González González
- R. Loewith
- David M. Sabatini
- E. Jacinto
- R. A. Saxton
- Kim Lewis
- M. Conrad
- K. Inoki
- Sovan Sarkar
- N. Hay
Papers from Michael N. Hall's perspective
Ordered by how strongly the researchers Michael N. Hall trusts endorse each paper. Same corpus, a different order for every researcher.
- TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
- TOR, a central controller of cell growth. (Cell)
- Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (Molecules and Cells)
- Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
- Raptor, a binding partner of target of rapamycin (TOR), mediates TOR action. (Cell)
- Tsc tumour suppressor proteins antagonize amino-acid–TOR signalling (Nature Cell Biology)
- TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
- The TSC1-2 tumor suppressor controls insulin–PI3K signaling via regulation of IRS proteins (Journal of Cell Biology)
- Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB. (Molecules and Cells)
- Mammalian cell size is controlled by mTOR and its downstream targets S6K1 and 4EBP1/eIF4E. (Genes & Development)
Their papers
- TOR signaling in growth and metabolism. (2006) — 6,014 citations
- Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (2002) — 1,926 citations
- TOR, a central controller of cell growth. (2000) — 2,036 citations
- Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive (2004) — 2,219 citations
- TOR signalling in bugs, brain and brawn (2003) — 595 citations
- Sch9 is a major target of TORC1 in Saccharomyces cerevisiae. (2007) — 824 citations
- In brief (2011) — 625 citations
- Rapamycin passes the torch: a new generation of mTOR inhibitors (2011) — 979 citations
- Target of Rapamycin (TOR) in Nutrient Signaling and Growth Control (2011) — 883 citations
- Making new contacts: the mTOR network in metabolism and signalling crosstalk (2014) — 1,043 citations
- Glutaminolysis activates Rag-mTORC1 signaling. (2012) — 689 citations
- Nutrient sensing and TOR signaling in yeast and mammals (2017) — 670 citations
- mTOR signalling and cellular metabolism are mutual determinants in cancer (2018) — 972 citations
- Cell growth : control of cell size (2004) — 134 citations