Y. Sancak

Molecular Biology · 2007–2013

A researcher in the Science corpus: 8 papers; trusts 4 researchers and is trusted by 11 researchers here, as inferred from the citation graph.

Subfields

Whom Y. Sancak trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Dos D. Sarbassov
  2. David M. Sabatini
  3. D. Guertin
  4. Siraj M. Ali
  5. M. Laplante
  6. K. Inoki
  7. C. Thoreen
  8. E. Eskelinen
  9. Timothy R. Peterson
  10. B. Manning

Who trusts Y. Sancak overall

  1. David M. Sabatini
  2. L. Bar-Peled
  3. M. Laplante
  4. R. A. Saxton
  5. Asier González González
  6. Michael N. Hall
  7. Roberto Zoncu
  8. Lynne Chantranupong
  9. Young Chul Kim
  10. K. Guan

Papers from Y. Sancak's perspective

Ordered by how strongly the researchers Y. Sancak trusts endorse each paper. Same corpus, a different order for every researcher.

  1. TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
  2. Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
  3. mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery. (Cell)
  4. Rictor, a novel binding partner of mTOR, defines a rapamycin-insensitive and raptor-independent pathway that regulates the cytoskeleton. (Current Biology)
  5. Phosphorylation and functional inactivation of TSC2 by Erk implications for tuberous sclerosis and cancer pathogenesis. (Cell)
  6. Tumor-promoting phorbol esters and activated Ras inactivate the tuberous sclerosis tumor suppressor complex via p90 ribosomal S6 kinase. (Proceedings of the National Academy of Sciences of the United States of America)
  7. TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
  8. Tsc tumour suppressor proteins antagonize amino-acid–TOR signalling (Nature Cell Biology)
  9. Raptor, a binding partner of target of rapamycin (TOR), mediates TOR action. (Cell)
  10. Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB. (Molecules and Cells)

Their papers