K. Inoki
Molecular Biology · 2002–2021
A researcher in the Science corpus: 11 papers; trusts 15 researchers and is trusted by 43 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 236 citations into it; #628 of 17,773 by trust here, #1,004 by citations
- Epidemiology — 145 citations into it
- Physiology — 89 citations into it; #110 of 2,276 by trust here, #157 by citations
- Surgery — 25 citations into it
- Ecology, Evolution, Behavior and Systematics — 5 citations into it
Whom K. Inoki trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Hardie
- D. Fingar
- Christopher Potter
- B. Manning
- S. Hawley
- Dos D. Sarbassov
- E. Jacinto
- J. Blenis
- Laura Pedraza
- R. Loewith
Who trusts K. Inoki overall
- David M. Sabatini
- M. Laplante
- Asier González González
- R. Shaw
- D. Hardie
- B. Manning
- Michael N. Hall
- K. Guan
- Young Chul Kim
- A. Budanov
Papers from K. Inoki's perspective
Ordered by how strongly the researchers K. Inoki trusts endorse each paper. Same corpus, a different order for every researcher.
- TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
- Tsc tumour suppressor proteins antagonize amino-acid–TOR signalling (Nature Cell Biology)
- Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (Molecules and Cells)
- mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery. (Cell)
- Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
- TOR, a central controller of cell growth. (Cell)
- TOR signaling in growth and metabolism. (Cell)
- Mammalian cell size is controlled by mTOR and its downstream targets S6K1 and 4EBP1/eIF4E. (Genes & Development)
- TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
- Role of AMP-activated protein kinase in mechanism of metformin action. (Journal of Clinical Investigation)
Their papers
- TSC2 mediates cellular energy response to control cell growth and survival. (2003) — 3,956 citations
- TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (2002) — 3,197 citations
- Rheb GTPase is a direct target of TSC2 GAP activity and regulates mTOR signaling. (2003) — 1,954 citations
- Regulation of the TSC pathway by LKB1: evidence of a molecular link between tuberous sclerosis complex and Peutz-Jeghers syndrome. (2004) — 595 citations
- Essential function of TORC2 in PKC and Akt turn motif phosphorylation, maturation and signalling (2008) — 629 citations
- TSC2 integrates Wnt and energy signals via a coordinated phosphorylation by AMPK and GSK3 to regulate cell growth. (2006) — 1,421 citations
- AMPK and mTOR in cellular energy homeostasis and drug targets. (2012) — 780 citations
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 (2021) — 3,841 citations
- ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS (2010) — 712 citations
- Guidelines for the use and interpretation of assays for monitoring autophagy (2012) — 4,856 citations
- Dysregulation of the TSC-mTOR pathway in human disease (2004) — 1,008 citations