J. Martinou

Molecular Biology · 2000–2018

A researcher in the Science corpus: 13 papers; trusts 7 researchers and is trusted by 21 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 364 citations into it; #2,268 of 17,773 by trust here, #1,368 by citations
• Clinical Biochemistry — 20 citations into it; #151 of 177 by trust here, #67 by citations
• Cellular and Molecular Neuroscience — 11 citations into it

Whom J. Martinou trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. S. Desagher
2. J. Nunnari
3. J. Chipuk
4. Douglas R. Green
5. G. Salvesen
6. B. Westermann
7. Guido Kroemer
8. R. Youle
9. T. Kuwana
10. M. Karbowski

Who trusts J. Martinou overall

1. D. C. Chan
2. Jerry M. Adams
3. A. Halestrap
4. R. Youle
5. M. Fricker
6. J. Nunnari
7. Aida Peña‐Blanco
8. M. Karbowski
9. Douglas R. Green
10. Wen Chen

Papers from J. Martinou's perspective

Ordered by how strongly the researchers J. Martinou trusts endorse each paper. Same corpus, a different order for every researcher.

1. Role of Bax and Bak in mitochondrial morphogenesis (Nature)
2. Mitochondrial rhomboid PARL regulates cytochrome c release during apoptosis via OPA1-dependent cristae remodeling. (Cell)
3. OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion. (Cell)
4. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (Molecules and Cells)
5. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
6. A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis. (Developmental Cell)
7. Mitochondria and cell death: outer membrane permeabilization and beyond (Nature reviews. Molecular cell biology)
8. hFis1, a Novel Component of the Mammalian Mitochondrial Fission Machinery* (Journal of Biological Chemistry)
9. Chemical inhibition of the mitochondrial division dynamin reveals its role in Bax/Bak-dependent mitochondrial outer membrane permeabilization. (Developmental Cell)
10. Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (Cancer Cell)

Their papers

• Bid Induces the Oligomerization and Insertion of Bax into the Outer Mitochondrial Membrane (2000) — 1,237 citations
• hFis1, a Novel Component of the Mammalian Mitochondrial Fission Machinery* (2003) — 670 citations
• MLKL compromises plasma membrane integrity by binding to phosphatidylinositol phosphates. (2014) — 783 citations
• SLP‐2 is required for stress‐induced mitochondrial hyperfusion (2009) — 791 citations
• Mitochondria as the central control point of apoptosis. (2000) — 1,987 citations
• Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 (2018) — 5,732 citations
• Essential versus accessory aspects of cell death: recommendations of the NCCD 2015 (2014) — 970 citations
• Nitric oxide‐induced mitochondrial fission is regulated by dynamin‐related GTPases in neurons (2006) — 681 citations
• Bax Is Present as a High Molecular Weight Oligomer/Complex in the Mitochondrial Membrane of Apoptotic Cells* (2001) — 728 citations
• Breaking the mitochondrial barrier (2001) — 1,012 citations
• Identification and Functional Expression of the Mitochondrial Pyruvate Carrier (2012) — 719 citations
• Mitochondria in Apoptosis: Bcl-2 family Members and Mitochondrial Dynamics (2011) — 1,375 citations
• Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria. (2000) — 658 citations