M. Fricker
Molecular Biology · 2018
A researcher in the Science corpus: 1 paper; trusts 8 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 72 citations into it
- Physiology — 34 citations into it
- Epidemiology — 32 citations into it
- Neurology — 24 citations into it
- Cell Biology — 17 citations into it
- Pulmonary and Respiratory Medicine — 7 citations into it
- Nutrition and Dietetics — 6 citations into it
Whom M. Fricker trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- W. Yang
- Y. Ohsumi
- E. Mandelkow
- Brent R. Stockwell
- S. Desagher
- Yang Liu
- Jianjin Shi
- S. Orrenius
- T. Lai
- Beth Levine
Papers from M. Fricker's perspective
Ordered by how strongly the researchers M. Fricker trusts endorse each paper. Same corpus, a different order for every researcher.
- Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (Cancer Cell)
- Ferroptosis: Death by Lipid Peroxidation. (Trends in Cell Biology)
- Cell death by mitotic catastrophe: a molecular definition (Oncogene)
- The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism. (Immunity)
- Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function. (Molecules and Cells)
- Ablation of ferroptosis regulator glutathione peroxidase 4 in forebrain neurons promotes cognitive impairment and neurodegeneration (Redox Biology)
- Cleavage of DFNA5 by caspase-3 during apoptosis mediates progression to secondary necrotic/pyroptotic cell death (Nature Communications)
- Pyroptosis: Gasdermin-Mediated Programmed Necrotic Cell Death. (TIBS -Trends in Biochemical Sciences. Regular ed)
- Bcl-2 antiapoptotic proteins inhibit Beclin 1-dependent autophagy. (Cell)
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
Their papers
- Neuronal Cell Death. (2018) — 1,023 citations