Breaking the mitochondrial barrier
By J. Martinou, Douglas R. Green. Nature reviews. Molecular cell biology, 2001
1,012 citations recorded by the source corpus.
Subfields: Molecular Biology
View Paper: https://www.semanticscholar.org/paper/95f0d34558f8c9dac3374dae01ce678cac5a103c
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Citations in the corpus
- Anoikis: an emerging hallmark in health and diseases (2012, background): “In the intrinsic pathway, caspase activation occurs as a consequence of mitochondrial permeabilization due to formation of oligomers in the outer mitochondrial membrane (OMM) by the pro-apoptotic proteins of the Bcl-2 family, thereby creating channels and causing membrane permeabilization [7,8].”
- Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation. (2003, background): “The balance between pro- and antiapoptotic Bcl-2 family members determines the mitochondrial response to apoptotic stimuli (Gross et al. 1999; Martinou and Green 2001).”
- Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection. (2004, background): “distinction between apoptosis and necrosis is that the former requires ATP whilst the latter occurs in its absence [24,25].”
- Apoptosis A Link between Cancer Genetics and Chemotherapy (2002, background): “…theability of cells to evade apoptosis is also an essential outer membrane, and/or by regulating the opening and“hallmark of cancer” (Hanahan and Weinberg, 2000). closing of the permeability transition pore remains theComponents of Apoptotic Pathways topic of much debate (Martinou and Green, 2001).”
- The permeability transition pore complex: another view. (2002, neutral): “These are induced by pro-apoptotic proteins such as BAX and Bid [3] [5].”
- The changing faces of glutathione, a cellular protagonist. (2003, background): “The rupture of the physical barrier (OMM) that limits their release into the cytosol constitutes a point-of-noreturn in cell death (Martinou and Green, 2001; Kroemer et al., 2007).”