J. Nunnari

Molecular Biology · 2005–2014

A researcher in the Science corpus: 6 papers; trusts 17 researchers and is trusted by 38 researchers here, as inferred from the citation graph.

Subfields

Whom J. Nunnari trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Hsiuchen Chen
  2. M. Karbowski
  3. D. C. Chan
  4. R. Youle
  5. Derek P. Narendra
  6. S. Cipolat
  7. Xinnan Wang
  8. Naotada Ishihara
  9. J. Chipuk
  10. M. Murphy

Who trusts J. Nunnari overall

  1. B. Westermann
  2. Xieyang Xu
  3. Hsiuchen Chen
  4. A. M. van der Bliek
  5. R. Youle
  6. J. Martinou
  7. Yan Pang
  8. S. Ferguson
  9. Ghazaleh Ashrafi
  10. L. Chernomordik

Papers from J. Nunnari's perspective

Ordered by how strongly the researchers J. Nunnari trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  2. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  3. Mitochondrial rhomboid PARL regulates cytochrome c release during apoptosis via OPA1-dependent cristae remodeling. (Cell)
  4. Roles of the mammalian mitochondrial fission and fusion mediators Fis1, Drp1, and Opa1 in apoptosis. (Molecular Biology of the Cell)
  5. The PINK1/Parkin pathway regulates mitochondrial morphology (Proceedings of the National Academy of Sciences of the United States of America)
  6. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
  7. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  8. Role of Bax and Bak in mitochondrial morphogenesis (Nature)
  9. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
  10. OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28 (Nature Genetics)

Their papers