Aida Peña‐Blanco
Molecular Biology · 2018
A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 30 citations into it
Whom Aida Peña‐Blanco trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Aida Peña‐Blanco's perspective
Ordered by how strongly the researchers Aida Peña‐Blanco trusts endorse each paper. Same corpus, a different order for every researcher.
- A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis. (Developmental Cell)
- Roles of the mammalian mitochondrial fission and fusion mediators Fis1, Drp1, and Opa1 in apoptosis. (Molecular Biology of the Cell)
- BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly. (Molecules and Cells)
- Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members. (Cancer Cell)
- Mitochondrial fission factor Drp1 is essential for embryonic development and synapse formation in mice (Nature Cell Biology)
- Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosis (Journal of Cell Biology)
- Membrane binding by tBid initiates an ordered series of events culminating in membrane permeabilization by Bax. (Cell)
- BAX Activation is Initiated at a Novel Interaction Site (Nature)
- The BCL-2 protein family: opposing activities that mediate cell death (Nature reviews. Molecular cell biology)
- Structure of Bax: coregulation of dimer formation and intracellular localization. (Cell)
Their papers
- Bax, Bak and beyond — mitochondrial performance in apoptosis (2018) — 834 citations