Jerry M. Adams

Molecular Biology · 2001–2013

A researcher in the Science corpus: 16 papers; trusts 40 researchers and is trusted by 32 researchers here, as inferred from the citation graph.

Subfields

Molecular Biology — 269 citations into it; #1,106 of 17,773 by trust here, #1,015 by citations
Oncology — 66 citations into it; #80 of 1,698 by trust here, #187 by citations
Immunology — 46 citations into it; #803 of 2,160 by trust here, #1,169 by citations
Epidemiology — 37 citations into it; #680 of 2,148 by trust here, #998 by citations
Cell Biology — 29 citations into it
Biotechnology — 17 citations into it

Whom Jerry M. Adams trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

Who trusts Jerry M. Adams overall

Papers from Jerry M. Adams's perspective

Ordered by how strongly the researchers Jerry M. Adams trusts endorse each paper. Same corpus, a different order for every researcher.

Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis. (Science)
Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (Cancer Cell)
Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (Molecules and Cells)
Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins. (Cell)
BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis. (Molecules and Cells)
BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak. (Genes & Development)
Endonuclease G is an apoptotic DNase when released from mitochondria (Nature)
Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation. (Genes & Development)
The expanding role of mitochondria in apoptosis. (Genes & Development)

Their papers

Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome (2002) — 596 citations
Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function. (2005) — 1,894 citations
Control of apoptosis by the BCL-2 protein family: implications for physiology and therapy (2013) — 3,023 citations
The Bcl2 family: regulators of the cellular life-or-death switch (2002) — 4,132 citations
BH3-only Bcl-2 family member Bim is required for apoptosis of autoreactive thymocytes (2002) — 847 citations
Ways of dying: multiple pathways to apoptosis. (2003) — 839 citations
The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralized. (2006) — 1,255 citations
p53- and Drug-Induced Apoptotic Responses Mediated by BH3-Only Proteins Puma and Noxa (2003) — 1,411 citations
Life in the balance: how BH3-only proteins induce apoptosis. (2005) — 796 citations
Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins. (2005) — 1,362 citations
Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak (2007) — 1,236 citations
The Bcl-2 apoptotic switch in cancer development and therapy (2007) — 2,202 citations
The Bcl-2 family: roles in cell survival and oncogenesis (2003) — 1,528 citations
Bcl-2-regulated apoptosis: mechanism and therapeutic potential. (2007) — 681 citations
Life-or-death decisions by the Bcl-2 protein family. (2001) — 943 citations
Tumor Growth Need Not Be Driven by Rare Cancer Stem Cells (2007) — 885 citations