J. Krueger
Immunology · 2002–2018
A researcher in the Science corpus: 15 papers; trusts 2 researchers and is trusted by 7 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 152 citations into it; #665 of 2,160 by trust here, #481 by citations
- Dermatology — 69 citations into it; #44 of 119 by trust here, #9 by citations
Whom J. Krueger trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. Krueger overall
Papers from J. Krueger's perspective
Ordered by how strongly the researchers J. Krueger trusts endorse each paper. Same corpus, a different order for every researcher.
- Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
- Psoriasis vulgaris lesions contain discrete populations of Th1 and Th17 T cells. (Journal of Investigative Dermatology)
- Development, cytokine profile and function of human interleukin 17–producing helper T cells (Nature Immunology)
- A human interleukin-12/23 monoclonal antibody for the treatment of psoriasis. (New England Journal of Medicine)
- TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
- Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells (Nature Immunology)
- Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain (Nature)
- IL-17 family cytokines and the expanding diversity of effector T cell lineages. (Annual Review of Immunology)
- IL-22 increases the innate immunity of tissues. (Immunity)
- Plasmacytoid dendritic cells sense self-DNA coupled with antimicrobial peptide (Nature)
Their papers
- Interleukin 23 in the skin: role in psoriasis pathogenesis and selective interleukin 23 blockade as treatment (2018) — 127 citations
- Survival of tissue-resident memory T cells requires exogenous lipid uptake and metabolism (2017) — 662 citations
- Psoriasis Pathogenesis and the Development of Novel, Targeted Immune Therapies (2017) — 867 citations
- Immunology of Psoriasis (2014) — 1,647 citations
- Brodalumab, an anti-interleukin-17-receptor antibody for psoriasis. (2012) — 954 citations
- Progressive activation of Th2/Th22 cytokines and selective epidermal proteins characterizes acute and chronic atopic dermatitis (2012) — 900 citations
- Integrative responses to IL-17 and TNF-α in human keratinocytes account for key inflammatory pathogenic circuits in psoriasis. (2011) — 687 citations
- Psoriasis and systemic inflammatory diseases: potential mechanistic links between skin disease and co-morbid conditions. (2010) — 731 citations
- IL-22 producing “T22” T-cells account for up-regulated IL-22 in atopic dermatitis (AD), despite reduced IL-17 producing Th17 T-cells (2009) — 613 citations
- Th17 cytokines interleukin (IL)‐17 and IL‐22 modulate distinct inflammatory and keratinocyte‐response pathways (2008) — 861 citations
- Psoriasis vulgaris lesions contain discrete populations of Th1 and Th17 T cells. (2008) — 1,116 citations
- Amelioration of epidermal hyperplasia by TNF inhibition is associated with reduced Th17 responses (2007) — 650 citations
- Pathogenesis and therapy of psoriasis (2007) — 1,810 citations
- Increased Expression of Interleukin 23 p19 and p40 in Lesional Skin of Patients with Psoriasis Vulgaris (2004) — 982 citations
- The immunologic basis for the treatment of psoriasis with new biologic agents. (2002) — 646 citations