J. O’Shea

Immunology · Immunity · 2001–2017

A researcher in the Science corpus: 30 papers; trusts 15 researchers and is trusted by 21 researchers here, as inferred from the citation graph.

Subfields

Whom J. O’Shea trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Warren S. Alexander
  2. A. Laurence
  3. Y. Kanno
  4. Elaine R. Mardis
  5. L. Platanias
  6. K. Shuai
  7. A. Yoshimura
  8. Liang Zhou
  9. James E. Darnell
  10. S. Holland

Who trusts J. O’Shea overall

  1. W. Liao
  2. Ting Liu
  3. Shane Crotty
  4. D. Mosser
  5. M. Karin
  6. H. Spits
  7. Jian-Xin Lin
  8. Shao-Cong Sun
  9. Y. Kanno
  10. Lingyun Zhang

Papers from J. O’Shea's perspective

Ordered by how strongly the researchers J. O’Shea trusts endorse each paper. Same corpus, a different order for every researcher.

  1. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  2. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
  3. Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
  4. Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)
  5. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
  6. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
  7. Essential autocrine regulation by IL-21 in the generation of inflammatory T cells (Nature)
  8. IL-17 family cytokines and the expanding diversity of effector T cell lineages. (Annual Review of Immunology)
  9. The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins (Nature)
  10. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)

Their papers