Shao-Cong Sun

Immunology · 2000–2017

A researcher in the Science corpus: 11 papers; trusts 20 researchers and is trusted by 15 researchers here, as inferred from the citation graph.

Subfields

• Immunology — 104 citations into it; #666 of 2,160 by trust here, #503 by citations
• Cancer Research — 68 citations into it; #161 of 1,780 by trust here, #236 by citations
• Molecular Biology — 52 citations into it; #3,729 of 17,773 by trust here, #4,020 by citations
• Oncology — 43 citations into it; #666 of 1,698 by trust here, #1,007 by citations
• Epidemiology — 9 citations into it

Whom Shao-Cong Sun trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Sivakumar Vallabhapurapu
2. R. Mebius
3. M. Hayden
4. M. Karin
5. S. Ghosh
6. C. Dong
7. J. O’Shea
8. M. Croft
9. D. Finley
10. A. Culyer

Who trusts Shao-Cong Sun overall

1. N. Perkins
2. M. Hayden
3. M. Park
4. Bastian Hoesel
5. Hui Yu
6. S. Ghosh
7. M. Karin
8. Ruey-Horng Shih
9. Joseph A. DiDonato
10. Qian Zhang

Papers from Shao-Cong Sun's perspective

Ordered by how strongly the researchers Shao-Cong Sun trusts endorse each paper. Same corpus, a different order for every researcher.

1. Missing pieces in the NF-kappaB puzzle. (Cell)
2. Nonredundant and complementary functions of TRAF2 and TRAF3 in a ubiquitination cascade that activates NIK-dependent alternative NF-κB signaling (Nature Immunology)
3. Non-transcriptional Priming and Deubiquitination Regulate NLRP3 Inflammasome Activation* (Journal of Biological Chemistry)
4. Deubiquitination of NLRP3 by BRCC3 critically regulates inflammasome activity. (Molecules and Cells)
5. The mTOR kinase differentially regulates effector and regulatory T cell lineage commitment. (Immunity)
6. Regulation of inflammasome activation (Immunological Reviews)
7. Activation by IKKα of a Second, Evolutionary Conserved, NF-κB Signaling Pathway (Science)
8. Epithelial NEMO links innate immunity to chronic intestinal inflammation (Nature)
9. cIAP1 and cIAP2 facilitate cancer cell survival by functioning as E3 ligases that promote RIP1 ubiquitination. (Molecules and Cells)
10. The CD28 signaling pathway regulates glucose metabolism. (Immunity)

Their papers

• NF-κB-Inducing Kinase Regulates the Processing of NF-κB2 p100 (2001) — 787 citations
• Induction of p100 Processing by NF-κB-inducing Kinase Involves Docking IκB Kinase α (IKKα) to p100 and IKKα-mediated Phosphorylation* (2004) — 271 citations
• Non-canonical NF-κB signaling pathway (2011) — 1,150 citations
• The noncanonical NF‐κB pathway (2012) — 623 citations
• β-TrCP binding and processing of NF-κB2/p100 involve its phosphorylation at serines 866 and 870 (2006) — 107 citations
• The non-canonical NF-κB pathway in immunity and inflammation (2017) — 1,697 citations
• NF-κB signaling in inflammation (2017) — 7,599 citations
• Potentiating the antitumour response of CD8+ T cells by modulating cholesterol metabolism (2016) — 892 citations
• Inflammatory T cell responses rely on amino acid transporter ASCT2 facilitation of glutamine uptake and mTORC1 kinase activation (2014) — 786 citations
• Activation by IKKα of a Second, Evolutionary Conserved, NF-κB Signaling Pathway (2001) — 1,312 citations
• Domain-specific Interaction with the IκB Kinase (IKK) Regulatory Subunit IKKγ Is an Essential Step in Tax-mediated Activation of IKK* (2000) — 99 citations