Y. Kanno
Immunology · 2004–2017
A researcher in the Science corpus: 11 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 179 citations into it; #1,349 of 2,160 by trust here, #235 by citations
- Oncology — 59 citations into it; #1,666 of 1,698 by trust here, #567 by citations
Whom Y. Kanno trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Y. Kanno overall
Papers from Y. Kanno's perspective
Ordered by how strongly the researchers Y. Kanno trusts endorse each paper. Same corpus, a different order for every researcher.
- IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
- Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
- Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
- Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
- Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
- Essential autocrine regulation by IL-21 in the generation of inflammatory T cells (Nature)
- Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)
- The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins (Nature)
- STAT3 Regulates Cytokine-mediated Generation of Inflammatory Helper T Cells* (Journal of Biological Chemistry)
Their papers
- JAK inhibition as a therapeutic strategy for immune and inflammatory diseases (2017) — 1,075 citations
- Mechanisms and consequences of Jak-STAT signaling in the immune system (2017) — 1,064 citations
- Opposing regulation of the Il17 locus through direct, reciprocal actions of STAT3 and STAT5 (2011) — 595 citations
- Generation of Pathogenic Th17 Cells in the Absence of TGF-β Signaling (2010) — 1,234 citations
- Diverse Targets of the Transcription Factor STAT3 Contribute to T Cell Pathogenicity and Homeostasis (2010) — 740 citations
- Global mapping of H3K4me3 and H3K27me3 reveals specificity and plasticity in lineage fate determination of differentiating CD4+ T cells. (2009) — 1,191 citations
- Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome (2008) — 727 citations
- Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation. (2007) — 1,600 citations
- Nonredundant roles for Stat5a/b in directly regulating Foxp3. (2007) — 610 citations
- Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells. (2006) — 712 citations
- Signaling by IL‐12 and IL‐23 and the immunoregulatory roles of STAT4 (2004) — 608 citations