A. Laurence

Immunology · 2006–2015

A researcher in the Science corpus: 13 papers; trusts 1 researcher and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

• Immunology — 204 citations into it; #730 of 2,160 by trust here, #168 by citations
• Oncology — 48 citations into it; #1,588 of 1,698 by trust here, #461 by citations

Whom A. Laurence trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Paul R. Mangan
2. L. Harrington
3. M. Veldhoen
4. Y. Kanno
5. E. Bettelli

Who trusts A. Laurence overall

1. J. O’Shea
2. T. Malek
3. Jinfang Zhu
4. A. Jetten
5. T. Hirano
6. T. Korn
7. E. Bettelli
8. C. Dong
9. R. Nurieva
10. Liang Zhou

Papers from A. Laurence's perspective

Ordered by how strongly the researchers A. Laurence trusts endorse each paper. Same corpus, a different order for every researcher.

1. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
2. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
3. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
4. Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
5. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
6. Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
7. Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12.
8. Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
9. IL-27, a heterodimeric cytokine composed of EBI3 and p28 protein, induces proliferation of naive CD4+ T cells.
10. Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)

Their papers

• The JAK-STAT Pathway: Impact on Human Disease and Therapeutic Intervention (2015) — 1,413 citations
• Opposing regulation of the Il17 locus through direct, reciprocal actions of STAT3 and STAT5 (2011) — 595 citations
• Generation of Pathogenic Th17 Cells in the Absence of TGF-β Signaling (2010) — 1,234 citations
• Diverse Targets of the Transcription Factor STAT3 Contribute to T Cell Pathogenicity and Homeostasis (2010) — 740 citations
• Janus kinases in immune cell signaling (2009) — 1,081 citations
• Interleukin 23 receptor is essential for terminal differentiation of effector T helper type 17 cells in vivo (2009) — 1,081 citations
• Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome (2008) — 727 citations
• Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation. (2007) — 1,600 citations
• IL-21 Is Produced by Th17 Cells and Drives IL-17 Production in a STAT3-dependent Manner* (2007) — 693 citations
• Interleukins 27 and 6 induce STAT3-mediated T cell production of interleukin 10 (2007) — 878 citations
• Nonredundant roles for Stat5a/b in directly regulating Foxp3. (2007) — 610 citations
• Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (2006) — 1,042 citations
• Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells. (2006) — 712 citations