C. Lewis

Immunology · Cancer Research · 2004–2013

A researcher in the Science corpus: 4 papers; trusts 8 researchers and is trusted by 17 researchers here, as inferred from the citation graph.

Subfields

Whom C. Lewis trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. J. Pollard
  2. A. Mantovani
  3. Elaine Lin
  4. Craig Murdoch
  5. Noam Brown
  6. J. Wyckoff
  7. Michele De Palma
  8. G. Bergers

Who trusts C. Lewis overall

  1. M. Egeblad
  2. D. Hanahan
  3. Y. Mao
  4. Lisa M. Coussens
  5. J. Pollard
  6. A. Mantovani
  7. E. Nakasone
  8. M. Munder
  9. T. Seyfried
  10. J. Joyce

Papers from C. Lewis's perspective

Ordered by how strongly the researchers C. Lewis trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Macrophage Diversity Enhances Tumor Progression and Metastasis (Cell)
  2. Macrophages regulate the angiogenic switch in a mouse model of breast cancer. (Cancer Research)
  3. Macrophage polarization: tumor-associated macrophages as a paradigm for polarized M2 mononuclear phagocytes. (Trends in immunology)
  4. Mechanisms regulating the recruitment of macrophages into hypoxic areas of tumors and other ischemic tissues. (Blood)
  5. Expansion of myeloid immune suppressor Gr+CD11b+ cells in tumor-bearing host directly promotes tumor angiogenesis. (Cancer Cell)
  6. The role of tumour‐associated macrophages in tumour progression: implications for new anticancer therapies (Journal of Pathology)
  7. De novo carcinogenesis promoted by chronic inflammation is B lymphocyte dependent. (Cancer Cell)
  8. Tie2 identifies a hematopoietic lineage of proangiogenic monocytes required for tumor vessel formation and a mesenchymal population of pericyte progenitors. (Cancer Cell)
  9. Infiltrating neutrophils mediate the initial angiogenic switch in a mouse model of multistage carcinogenesis (Proceedings of the National Academy of Sciences of the United States of America)
  10. HIF1alpha induces the recruitment of bone marrow-derived vascular modulatory cells to regulate tumor angiogenesis and invasion. (Cancer Cell)

Their papers