Craig Murdoch

Immunology · 2000–2008

A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 8 researchers here, as inferred from the citation graph.

Subfields

Whom Craig Murdoch trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Noam Brown
  2. G. Bergers
  3. Elaine Lin
  4. A. Zlotnik
  5. J. Pollard

Who trusts Craig Murdoch overall

  1. D. Hanahan
  2. A. Mantovani
  3. M. Egeblad
  4. J. Pollard
  5. R. Gillitzer
  6. Alicia S. Chung
  7. T. Foster
  8. C. Lewis
  9. R. Weinberg
  10. J. Joyce

Papers from Craig Murdoch's perspective

Ordered by how strongly the researchers Craig Murdoch trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Expansion of myeloid immune suppressor Gr+CD11b+ cells in tumor-bearing host directly promotes tumor angiogenesis. (Cancer Cell)
  2. Matrix metalloproteinase-9 triggers the angiogenic switch during carcinogenesis (Nature Cell Biology)
  3. De novo carcinogenesis promoted by chronic inflammation is B lymphocyte dependent. (Cancer Cell)
  4. Macrophages regulate the angiogenic switch in a mouse model of breast cancer. (Cancer Research)
  5. Colony-Stimulating Factor 1 Promotes Progression of Mammary Tumors to Malignancy (Journal of Experimental Medicine)
  6. Infiltrating neutrophils mediate the initial angiogenic switch in a mouse model of multistage carcinogenesis (Proceedings of the National Academy of Sciences of the United States of America)
  7. A distinct and unique transcriptional program expressed by tumor-associated macrophages (defective NF-kappaB and enhanced IRF-3/STAT1 activation). (Blood)
  8. Mechanisms regulating the recruitment of macrophages into hypoxic areas of tumors and other ischemic tissues. (Blood)
  9. Identification of discrete tumor-induced myeloid-derived suppressor cell subpopulations with distinct T cell-suppressive activity. (Blood)
  10. Tie2 identifies a hematopoietic lineage of proangiogenic monocytes required for tumor vessel formation and a mesenchymal population of pericyte progenitors. (Cancer Cell)

Their papers