B. Leonard
Cellular and Molecular Neuroscience · Neuroscience and Biobehavioral Reviews · 2003–2012
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Cellular and Molecular Neuroscience — 13 citations into it
- Developmental Neuroscience — 12 citations into it
Whom B. Leonard trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts B. Leonard overall
Papers from B. Leonard's perspective
Ordered by how strongly the researchers B. Leonard trusts endorse each paper. Same corpus, a different order for every researcher.
- More hippocampal neurons in adult mice living in an enriched environment
- Glucocorticoid receptors in major depression: relevance to pathophysiology and treatment. (Biological Psychiatry)
- Depression: a case of neuronal life and death? (Biological Psychiatry)
- Chronic Antidepressant Treatment Increases Neurogenesis in Adult Rat Hippocampus
- Inflammatory Blockade Restores Adult Hippocampal Neurogenesis
- Inflammation is detrimental for neurogenesis in adult brain (Proceedings of the National Academy of Sciences of the United States of America)
- Hippocampal volume reduction in major depression. (American Journal of Psychiatry)
- Increased hippocampal BDNF immunoreactivity in subjects treated with antidepressant medication. (Biological Psychiatry)
- Neurogenesis in the adult human hippocampus
- Increased neurogenesis in a model of electroconvulsive therapy. (Biological Psychiatry)
Their papers
- The olfactory bulbectomised rat as a model of depression. (2005) — 682 citations
- Mechanistic explanations how cell-mediated immune activation, inflammation and oxidative and nitrosative stress pathways and their sequels and concomitants play a role in the pathophysiology of unipolar depression. (2012) — 781 citations
- The new '5-HT' hypothesis of depression: cell-mediated immune activation induces indoleamine 2,3-dioxygenase, which leads to lower plasma tryptophan and an increased synthesis of detrimental tryptophan catabolites (TRYCATs), both of which contribute to the onset of depression. (2011) — 665 citations
- A review of the relevance and validity of olfactory bulbectomy as a model of depression (2003) — 106 citations