Hsiuchen Chen

Molecular Biology · 2003–2016

A researcher in the Science corpus: 10 papers; trusts 14 researchers and is trusted by 40 researchers here, as inferred from the citation graph.

Subfields

Whom Hsiuchen Chen trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. D. C. Chan
  2. Scott A. Detmer
  3. S. Cipolat
  4. J. Nunnari
  5. Zhiyin Song
  6. A. Chomyn
  7. Naotada Ishihara
  8. C. Frezza
  9. Andrew J. Ewald
  10. Koji Okamoto

Who trusts Hsiuchen Chen overall

  1. D. C. Chan
  2. Angelika S. Rambold
  3. B. Westermann
  4. Zhiyin Song
  5. Xinglong Wang
  6. Prashant Mishra
  7. J. Nunnari
  8. Naotada Ishihara
  9. M. Liesa
  10. A. B. Knott

Papers from Hsiuchen Chen's perspective

Ordered by how strongly the researchers Hsiuchen Chen trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Disruption of Fusion Results in Mitochondrial Heterogeneity and Dysfunction* (Journal of Biological Chemistry)
  2. Dynamin-related protein Drp1 is required for mitochondrial division in mammalian cells. (Molecular Biology of the Cell)
  3. Roles of the mammalian mitochondrial fission and fusion mediators Fis1, Drp1, and Opa1 in apoptosis. (Molecular Biology of the Cell)
  4. Control of mitochondrial morphology by a human mitofusin. (Journal of Cell Science)
  5. Mitochondrial rhomboid PARL regulates cytochrome c release during apoptosis via OPA1-dependent cristae remodeling. (Cell)
  6. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  7. The Mitochondrial Protein hFis1 Regulates Mitochondrial Fission in Mammalian Cells through an Interaction with the Dynamin-Like Protein DLP1 (Molecular and Cellular Biology)
  8. Mitochondrial fusion and fission in mammals. (Annual Review of Cell and Developmental Biology)
  9. The PINK1/Parkin pathway regulates mitochondrial morphology (Proceedings of the National Academy of Sciences of the United States of America)
  10. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)

Their papers