Koji Okamoto

Molecular Biology · 2005–2021

A researcher in the Science corpus: 5 papers; trusts 8 researchers and is trusted by 7 researchers here, as inferred from the citation graph.

Subfields

Whom Koji Okamoto trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. J. Lemasters
  2. N. Mizushima
  3. Derek P. Narendra
  4. M. Karbowski

Who trusts Koji Okamoto overall

  1. Ghazaleh Ashrafi
  2. D. C. Chan
  3. W. Ding
  4. Thomas Schwarz
  5. Mashun Onishi
  6. B. Westermann
  7. Hsiuchen Chen
  8. Xiao-Ming Yin
  9. G. Twig
  10. R. Youle

Papers from Koji Okamoto's perspective

Ordered by how strongly the researchers Koji Okamoto trusts endorse each paper. Same corpus, a different order for every researcher.

  1. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  2. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  3. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  4. Nix is a selective autophagy receptor for mitochondrial clearance (EMBO Reports)
  5. LC3, GABARAP and GATE16 localize to autophagosomal membrane depending on form-II formation (Journal of Cell Science)
  6. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  7. Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy (Human Molecular Genetics)
  8. Essential role for Nix in autophagic maturation of erythroid cells (Nature)
  9. Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (Proceedings of the National Academy of Sciences of the United States of America)
  10. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)

Their papers