Rémi-Martin Laberge
Physiology · 2012–2026
A researcher in the Science corpus: 6 papers; trusts 5 researchers and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 115 citations into it; #1,310 of 2,276 by trust here, #499 by citations
- Molecular Biology — 95 citations into it; #13,223 of 17,773 by trust here, #8,089 by citations
- Immunology — 44 citations into it
Whom Rémi-Martin Laberge trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Jean-Philippe Coppé
- J. Campisi
- F. Rodier
- A. Krtolica
- S. Parrinello
- Adam Freund
- V. Krizhanovsky
- C. Patil
- J. Harborth
Who trusts Rémi-Martin Laberge overall
Papers from Rémi-Martin Laberge's perspective
Ordered by how strongly the researchers Rémi-Martin Laberge trusts endorse each paper. Same corpus, a different order for every researcher.
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (Nature Cell Biology)
- Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
- Oxygen sensitivity severely limits the replicative lifespan of murine fibroblasts (Nature Cell Biology)
- Cellular senescence in cancer and aging. (Cell)
- The Senescence-Associated Secretory Phenotype: The Dark Side of Tumor Suppression (Annual Review of Pathology)
- Lamin B1 loss is a senescence-associated biomarker (Molecular Biology of the Cell)
- Inflammatory Networks during Cellular Senescence: Causes and Consequences (Trends in Molecular Medicine)
Their papers
- Lamin B1 loss is a senescence-associated biomarker (2012) — 933 citations
- MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation (2015) — 1,061 citations
- An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA. (2026) — 1,614 citations
- Local clearance of senescent cells attenuates the development of post-traumatic osteoarthritis and creates a pro-regenerative environment (2017) — 1,334 citations
- Senescent cells: an emerging target for diseases of ageing (2017) — 1,101 citations
- Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice (2015) — 1,619 citations