V. Krizhanovsky
Physiology · 2006–2020
A researcher in the Science corpus: 8 papers; trusts 3 researchers and is trusted by 11 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 160 citations into it; #272 of 2,276 by trust here, #138 by citations
- Molecular Biology — 148 citations into it; #16,660 of 17,773 by trust here, #6,283 by citations
- Immunology — 63 citations into it; #1,342 of 2,160 by trust here, #990 by citations
- Aging — 25 citations into it
- Epidemiology — 20 citations into it
- Hepatology — 7 citations into it
Whom V. Krizhanovsky trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts V. Krizhanovsky overall
- J. Campisi
- Adam Freund
- F. Rodier
- Bennett G. Childs
- S. Friedman
- D. Baker
- T. Tchkonia
- T. Kuilman
- J. Puche
- C. Crewe
Papers from V. Krizhanovsky's perspective
Ordered by how strongly the researchers V. Krizhanovsky trusts endorse each paper. Same corpus, a different order for every researcher.
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
- Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
- Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (PLoS Biology)
- Living on a break: cellular senescence as a DNA-damage response (Nature Reviews. Cancer)
- Feedback between p21 and reactive oxygen production is necessary for cell senescence (Molecular Systems Biology)
- Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders (Nature)
- Liver fibrosis (Springer Seminars in Immunopathology)
Their papers
- Senescence is a developmental mechanism that contributes to embryonic growth and patterning. (2013) — 1,081 citations
- A novel role for high-mobility group a proteins in cellular senescence and heterochromatin formation. (2006) — 636 citations
- Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL (2016) — 887 citations
- Cellular senescence in ageing: from mechanisms to therapeutic opportunities (2020) — 1,893 citations
- Cellular Senescence: Defining a Path Forward. (2019) — 2,621 citations
- Non-cell-autonomous tumor suppression by p53 (2013) — 749 citations
- Senescence of activated stellate cells limits liver fibrosis (2008) — 1,898 citations
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (2007) — 2,392 citations