C. Patil

Molecular Biology · 2001–2015

A researcher in the Science corpus: 6 papers; trusts 6 researchers and is trusted by 13 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 85 citations into it; #2,875 of 17,773 by trust here, #1,821 by citations
• Physiology — 72 citations into it; #638 of 2,276 by trust here, #323 by citations
• Immunology — 39 citations into it; #1,249 of 2,160 by trust here, #641 by citations
• Cancer Research — 26 citations into it; #1,358 of 1,780 by trust here, #727 by citations

Whom C. Patil trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. Jean-Philippe Coppé
2. J. Campisi
3. A. Krtolica
4. F. Rodier
5. S. Parrinello
6. P. Desprez
7. M. Karin

Who trusts C. Patil overall

1. J. Campisi
2. R. Kaufman
3. Adam Freund
4. D. Todd
5. Matthew H. Brush
6. Ann-Hwee Lee
7. F. Rodier
8. J. Malhotra
9. M. Demaria
10. Douglas Weiser

Papers from C. Patil's perspective

Ordered by how strongly the researchers C. Patil trusts endorse each paper. Same corpus, a different order for every researcher.

1. Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
2. Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
3. Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
4. Cellular Senescence in Aging Primates (Science)
5. Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
6. Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (Nature Cell Biology)
7. Putting tumours in context (Nature Reviews. Cancer)
8. Oncogenic BRAF induces senescence and apoptosis through pathways mediated by the secreted protein IGFBP7. (Cell)
9. Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints (Nature)
10. Accumulation of senescent cells in mitotic tissue of aging primates. (Mechanisms of Ageing and Development)

Their papers

• Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (2008) — 3,837 citations
• Intracellular signaling from the endoplasmic reticulum to the nucleus: the unfolded protein response in yeast and mammals. (2001) — 827 citations
• Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (2009) — 2,520 citations
• p38MAPK is a novel DNA damage response‐independent regulator of the senescence‐associated secretory phenotype (2011) — 983 citations
• MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation (2015) — 1,061 citations
• Expression of microRNA-146 suppresses NF-κB activity with reduction of metastatic potential in breast cancer cells (2008) — 659 citations