T. Ganz

Hematology · 2001–2019

A researcher in the Science corpus: 25 papers; trusts 15 researchers and is trusted by 47 researchers here, as inferred from the citation graph.

Subfields

Whom T. Ganz trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. E. Nemeth
  2. R. Hurrell
  3. Matthias W. Hentze
  4. S. Rivera
  5. C. Peyssonnaux
  6. Delphine Meynard
  7. Ruihong Wang
  8. M. Zimmermann
  9. Léon Kautz
  10. V. Gabayan

Who trusts T. Ganz overall

  1. E. Nemeth
  2. T. Peel
  3. V. Haase
  4. P. Elias
  5. J. Wish
  6. A. Segal
  7. Donat R. Spahn
  8. Y. Gordon
  9. G. Papanikolaou
  10. S. Rivera

Papers from T. Ganz's perspective

Ordered by how strongly the researchers T. Ganz trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Hepcidin, a Urinary Antimicrobial Peptide Synthesized in the Liver* (Journal of Biological Chemistry)
  2. A New Mouse Liver-specific Gene, Encoding a Protein Homologous to Human Antimicrobial Peptide Hepcidin, Is Overexpressed during Iron Overload* (Journal of Biological Chemistry)
  3. Hepcidin Regulates Cellular Iron Efflux by Binding to Ferroportin and Inducing Its Internalization (Science)
  4. IL-6 mediates hypoferremia of inflammation by inducing the synthesis of the iron regulatory hormone hepcidin. (Journal of Clinical Investigation)
  5. Mutant antimicrobial peptide hepcidin is associated with severe juvenile hemochromatosis (Nature Genetics)
  6. Bone morphogenetic protein signaling by hemojuvelin regulates hepcidin expression (Nature Genetics)
  7. Severe iron deficiency anemia in transgenic mice expressing liver hepcidin (Proceedings of the National Academy of Sciences of the United States of America)
  8. A role of SMAD4 in iron metabolism through the positive regulation of hepcidin expression. (Cell Metabolism)
  9. Lack of hepcidin gene expression and severe tissue iron overload in upstream stimulatory factor 2 (USF2) knockout mice (Proceedings of the National Academy of Sciences of the United States of America)
  10. Inappropriate expression of hepcidin is associated with iron refractory anemia: implications for the anemia of chronic disease. (Blood)

Their papers