M. Beal
Molecular Biology · 2002–2020
A researcher in the Science corpus: 15 papers; trusts 19 researchers and is trusted by 37 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 115 citations into it; #1,055 of 17,773 by trust here, #2,156 by citations
- Physiology — 82 citations into it; #97 of 2,276 by trust here, #458 by citations
- Cellular and Molecular Neuroscience — 67 citations into it; #723 of 2,998 by trust here, #516 by citations
- Neurology — 66 citations into it; #68 of 1,455 by trust here, #225 by citations
- Pharmacology — 10 citations into it
- Clinical Biochemistry — 9 citations into it; #43 of 177 by trust here, #34 by citations
- Computational Theory and Mathematics — 8 citations into it
Whom M. Beal trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Douglas C. Wallace
- Tao Lu
- D. Selkoe
- M. Mańczak
- George Perry
- Hsiuchen Chen
- Giselle P. Lim
- T. Nakagawa
Who trusts M. Beal overall
- U. Schlattner
- P. Reddy
- Zhichun Chen
- Peter F Surai
- Rui Wang
- M. Mańczak
- M. Tokarska-Schlattner
- S. Gandhi
- I. Dalle-Donne
- S. Barber
Papers from M. Beal's perspective
Ordered by how strongly the researchers M. Beal trusts endorse each paper. Same corpus, a different order for every researcher.
- Mitochondrial Abnormalities in Alzheimer's Disease (Journal of Neuroscience)
- A Mitochondrial Paradigm of Metabolic and Degenerative Diseases, Aging, and Cancer: A Dawn for Evolutionary Medicine (Annual Review of Genetics)
- Premature ageing in mice expressing defective mitochondrial DNA polymerase (Nature)
- Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
- Mitochondria take center stage in aging and neurodegeneration (Annals of Neurology)
- Increased Lipid Peroxidation Precedes Amyloid Plaque Formation in an Animal Model of Alzheimer Amyloidosis (Journal of Neuroscience)
- Pathways towards and away from Alzheimer's disease (Nature)
- Transcriptional regulatory circuits controlling mitochondrial biogenesis and function. (Genes & Development)
- Alzheimer's Disease Is a Synaptic Failure (Science)
- Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. (Human Molecular Genetics)
Their papers
- Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases (2006) — 6,263 citations
- Functional engraftment of human ES cell–derived dopaminergic neurons enriched by coculture with telomerase-immortalized midbrain astrocytes (2006) — 832 citations
- Mitochondria take center stage in aging and neurodegeneration (2005) — 966 citations
- Neural subtype specification of fertilization and nuclear transfer embryonic stem cells and application in parkinsonian mice (2003) — 666 citations
- Mitochondrial α-Ketoglutarate Dehydrogenase Complex Generates Reactive Oxygen Species (2004) — 611 citations
- Oxidatively modified proteins in aging and disease. (2002) — 803 citations
- Amyloid beta, mitochondrial dysfunction and synaptic damage: implications for cognitive decline in aging and Alzheimer's disease. (2008) — 961 citations
- Mutated Human SOD1 Causes Dysfunction of Oxidative Phosphorylation in Mitochondria of Transgenic Mice* (2002) — 631 citations
- Floor plate-derived dopamine neurons from hESCs efficiently engraft in animal models of PD (2011) — 1,716 citations
- Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis (2008) — 791 citations
- Mitochondrial Dysfunction in Neurodegenerative Diseases (2012) — 857 citations
- Intraneuronal Alzheimer abeta42 accumulates in multivesicular bodies and is associated with synaptic pathology. (2002) — 767 citations
- Brain energy rescue: an emerging therapeutic concept for neurodegenerative disorders of ageing (2020) — 833 citations
- Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (2006) — 839 citations
- Effects of coenzyme Q10 in early Parkinson disease: evidence of slowing of the functional decline. (2002) — 1,036 citations