M. Mańczak

Physiology · 2006–2011

A researcher in the Science corpus: 3 papers; trusts 3 researchers and is trusted by 8 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 54 citations into it; #549 of 2,276 by trust here, #986 by citations
• Molecular Biology — 44 citations into it; #6,578 of 17,773 by trust here, #8,280 by citations
• Pharmacology — 25 citations into it; #803 of 1,112 by trust here, #451 by citations
• Computational Theory and Mathematics — 8 citations into it

Whom M. Mańczak trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Selkoe
2. R. Kayed
3. M. Beal
4. P. Reddy
5. Mark P. Mattson
6. F. LaFerla
7. Xinglong Wang
8. Giselle P. Lim
9. George Perry
10. D. Walsh

Who trusts M. Mańczak overall

1. M. Calkins
2. Xieyang Xu
3. Michael T. Lin
4. P. Reddy
5. E. Tönnies
6. M. Beal
7. R. Swerdlow
8. M. H. Yan
9. Yan Pang
10. Xinglong Wang

Papers from M. Mańczak's perspective

Ordered by how strongly the researchers M. Mańczak trusts endorse each paper. Same corpus, a different order for every researcher.

1. Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
2. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
3. Mitochondrial Abnormalities in Alzheimer's Disease (Journal of Neuroscience)
4. Increased Lipid Peroxidation Precedes Amyloid Plaque Formation in an Animal Model of Alzheimer Amyloidosis (Journal of Neuroscience)
5. Pathways towards and away from Alzheimer's disease (Nature)
6. Alzheimer's Disease Is a Synaptic Failure (Science)
7. Mitochondrial Aβ: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease
8. Mitochondria take center stage in aging and neurodegeneration (Annals of Neurology)
9. Nitric oxide‐induced mitochondrial fission is regulated by dynamin‐related GTPases in neurons (EMBO Journal)
10. A "mitochondrial cascade hypothesis" for sporadic Alzheimer's disease. (Medical Hypotheses)

Their papers

• Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. (2006) — 1,143 citations
• Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage. (2011) — 783 citations
• Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease. (2011) — 598 citations