P. Reddy
Molecular Biology · Human Molecular Genetics · 2006–2022
A researcher in the Science corpus: 6 papers; trusts 8 researchers and is trusted by 8 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 123 citations into it; #5,893 of 17,773 by trust here, #6,893 by citations
- Physiology — 121 citations into it; #458 of 2,276 by trust here, #793 by citations
- Pharmacology — 32 citations into it; #1,087 of 1,112 by trust here, #451 by citations
- Cellular and Molecular Neuroscience — 22 citations into it
- Neurology — 18 citations into it
- Biochemistry — 17 citations into it
- Computational Theory and Mathematics — 11 citations into it
Whom P. Reddy trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Selkoe
- George Perry
- M. Beal
- Mark P. Mattson
- M. Mańczak
- F. LaFerla
- S. Oddo
- Michael T. Lin
- M. Murphy
- A. Abramov
Who trusts P. Reddy overall
Papers from P. Reddy's perspective
Ordered by how strongly the researchers P. Reddy trusts endorse each paper. Same corpus, a different order for every researcher.
- Mitochondrial Abnormalities in Alzheimer's Disease (Journal of Neuroscience)
- Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
- Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
- Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction. (Neuron)
- Alzheimer's Disease Is a Synaptic Failure (Science)
- Increased Lipid Peroxidation Precedes Amyloid Plaque Formation in an Animal Model of Alzheimer Amyloidosis (Journal of Neuroscience)
- Mitochondrial Aβ: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease
- Nitric oxide‐induced mitochondrial fission is regulated by dynamin‐related GTPases in neurons (EMBO Journal)
- Pathways towards and away from Alzheimer's disease (Nature)
- Mitochondria take center stage in aging and neurodegeneration (Annals of Neurology)
Their papers
- Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. (2006) — 1,143 citations
- Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage. (2011) — 783 citations
- Amyloid beta, mitochondrial dysfunction and synaptic damage: implications for cognitive decline in aging and Alzheimer's disease. (2008) — 961 citations
- Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease. (2011) — 598 citations
- Oxidative stress in the pathophysiology of type 2 diabetes and related complications: Current therapeutics strategies and future perspectives. (2022) — 599 citations
- Mitochondrial dysfunction and oxidative stress in metabolic disorders - A Step towards mitochondria based therapeutic strategies (2016) — 1,410 citations