Mitochondrial Dysfunction in Neurodegenerative Diseases

By A. Johri, M. Beal. Journal of Pharmacology and Experimental Therapeutics, 2012

857 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Neurology

View Paper: https://www.semanticscholar.org/paper/0fa7032234a3ec7b6d27ff3c5b3f648cdb6fbc0e

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. E. Trushina
2. E. Tönnies
3. Xia Zhang
4. Wei-Wei Chen
5. Wenjuan Huang
6. P. Maher
7. Jana Lewerenz

In Molecular Biology

1. E. Trushina
2. E. Tönnies
3. Xia Zhang
4. Wei-Wei Chen
5. Wenjuan Huang
6. P. Maher
7. Jana Lewerenz

In Neurology

1. E. Trushina
2. E. Tönnies
3. Xia Zhang
4. Wei-Wei Chen
5. Wenjuan Huang

Citations in the corpus

• Role of oxidative stress in Alzheimer's disease (2016, background): “Thus, mitochondria function as both the source and target of toxic ROS since mitochondrial dysfunction and oxidative stress are important in aging and neurodegenerative diseases, particularly AD (11,12).”
• Oxidative Stress, Synaptic Dysfunction, and Alzheimer’s Disease (2017, background): “Mitochondrial dysfunction has been well documented in AD [34, 35].”
• Pesticides and human chronic diseases: evidences, mechanisms, and perspectives. (2013, neutral): “, 2012) and neurodegenerative disorders (Johri and Beal, 2012).”
• Chronic Glutamate Toxicity in Neurodegenerative Diseases—What is the Evidence? (2015, background): “First, although compromised mitochondrial function has been repeatedly described in several neurodegenerative diseases (reviewed in Johri and Beal, 2012), the resulting impairments in energy supply are not nearly as severe as the energy failure in ischemic stroke.”