Joseph V. Bonventre
Nephrology · 2001–2019
A researcher in the Science corpus: 19 papers; trusts 5 researchers and is trusted by 22 researchers here, as inferred from the citation graph.
Subfields
- Nephrology — 82 citations into it; #4 of 92 by trust here, #13 by citations
- Molecular Biology — 79 citations into it; #2,810 of 17,773 by trust here, #4,078 by citations
- Genetics — 64 citations into it
- Pulmonary and Respiratory Medicine — 21 citations into it; #770 of 770 by trust here, #286 by citations
Whom Joseph V. Bonventre trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Joseph V. Bonventre overall
- Tatiana Y. Besschetnova
- V. Vaidya
- P. Devarajan
- D. Basile
- L. Chawla
- C. Brooks
- Melissa D. Anderson
- Jeremy S. Duffield
- M. Ferguson
- K. Makris
Papers from Joseph V. Bonventre's perspective
Ordered by how strongly the researchers Joseph V. Bonventre trusts endorse each paper. Same corpus, a different order for every researcher.
- Epithelial-mesenchymal transition and its implications for fibrosis. (Journal of Clinical Investigation)
- The definition, classification, and prognosis of chronic kidney disease: a KDIGO Controversies Conference report. (Kidney International)
- Intrinsic epithelial cells repair the kidney after injury. (Cell Stem Cell)
- Hospital-acquired renal insufficiency. (American Journal of Kidney Diseases)
- The support of human genetic evidence for approved drug indications (Nature Genetics)
- Cre recombinase: The universal reagent for genome tailoring (Genesis)
- Fibroblasts in kidney fibrosis emerge via endothelial-to-mesenchymal transition. (Journal of the American Society of Nephrology)
- Prevalence of chronic kidney disease and decreased kidney function in the adult US population: Third National Health and Nutrition Examination Survey. (American Journal of Kidney Diseases)
- Six2 defines and regulates a multipotent self-renewing nephron progenitor population throughout mammalian kidney development (Cell Stem Cell)
- Endothelial-to-mesenchymal transition contributes to cardiac fibrosis (Nature Medicine)
Their papers
- Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis. (2010) — 1,446 citations
- Intrinsic epithelial cells repair the kidney after injury. (2008) — 866 citations
- Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury (2010) — 1,270 citations
- Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells. (2005) — 597 citations
- Dedifferentiation and proliferation of surviving epithelial cells in acute renal failure. (2003) — 607 citations
- Kidney Injury Molecule-1 (KIM-1): a novel biomarker for human renal proximal tubule injury. (2002) — 1,890 citations
- Flow-enhanced vascularization and maturation of kidney organoids in vitro (2019) — 796 citations
- Global kidney health 2017 and beyond: a roadmap for closing gaps in care, research, and policy. (2017) — 787 citations
- Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD (2015) — 824 citations
- Modeling kidney disease with CRISPR-mutant kidney organoids derived from human pluripotent epiblast spheroids (2015) — 710 citations
- Cellular pathophysiology of ischemic acute kidney injury. (2011) — 1,900 citations
- Creatinine kinetics and the definition of acute kidney injury. (2009) — 634 citations
- Biomarkers of acute kidney injury. (2008) — 1,015 citations
- Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells. (2008) — 747 citations
- Urinary kidney injury molecule-1: a sensitive quantitative biomarker for early detection of kidney tubular injury. (2006) — 662 citations
- Acute kidney injury, mortality, length of stay, and costs in hospitalized patients. (2005) — 3,267 citations
- Ischemic acute renal failure: an inflammatory disease? (2004) — 831 citations
- Recent advances in the pathophysiology of ischemic acute renal failure. (2003) — 793 citations
- Interleukin-1β-mediated induction of Cox-2 in the CNS contributes to inflammatory pain hypersensitivity (2001) — 1,322 citations