Ischemic acute renal failure: an inflammatory disease?

By Joseph V. Bonventre, A. Zuk. Kidney International, 2004

831 citations recorded by the source corpus.

Subfields: Immunology, Nephrology

View Paper: https://www.semanticscholar.org/paper/2db8d90078ee0c366620be8dce4e63d9fe4b4afc

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. D. Basile
2. T. Sutton
3. Melissa D. Anderson
4. V. Vaidya
5. Joseph V. Bonventre

In Nephrology

1. T. Sutton
2. Melissa D. Anderson
3. D. Basile
4. V. Vaidya
5. M. Ferguson
6. C. Edelstein
7. Abdullah Ozkok
8. Joseph V. Bonventre

Citations in the corpus

• Pathophysiology of Acute Kidney Injury (2012, positive): “These factors act in concert to promote inflammation in a positive feedback loop promoting further kidney injury (63).”
• Pathophysiology of Cisplatin-Induced Acute Kidney Injury (2014, background): “The vascular endothelial injury is an important component of cisplatininduced AKI [166].”
• Biomarkers of acute kidney injury. (2008, background): “Thus, inflammation contributes in a critical way to the pathophysiology of AKI (16).”
• Administered mesenchymal stem cells protect against ischemic acute renal failure through differentiation-independent mechanisms. (2005, background): “Recently, the importance of disordered vascular function and inflammation in the overall pathophysiology of ARF has been increasingly recognized (4, 19).”