Cellular pathophysiology of ischemic acute kidney injury.

By Joseph V. Bonventre, Li Yang. Journal of Clinical Investigation, 2011

1,900 citations recorded by the source corpus.

Subfields: Nephrology

View Paper: https://www.semanticscholar.org/paper/e151a602d4ed7ddcd5390615e4a6fad3e979bb4c

How researchers see this paper

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In Nephrology

1. Jean-Louis Vincent
2. Sean M. Bagshaw
3. L. Chawla

Citations in the corpus

• Discovery and validation of cell cycle arrest biomarkers in human acute kidney injury (2013, background): “Furthermore, these molecules are known to be associated with mechanisms recently implicated in the pathogenesis of AKI [15-17].”
• A Unified Theory of Sepsis-Induced Acute Kidney Injury: Inflammation, microcirculatory dysfunction, bioenergetics and the tubular cell adaptation to injury (2014, neutral): “This seems to be associated with the expression in S1, but not in S2 epithelial cells of heme oxygenase 1 (HO-1) and Sirt1, both highly protective against oxidative damage (22, 79).”