D. Fingar

Molecular Biology · 2002–2012

A researcher in the Science corpus: 7 papers; trusts 6 researchers and is trusted by 18 researchers here, as inferred from the citation graph.

Subfields

Whom D. Fingar trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Lewis C. Cantley
  2. K. Inoki
  3. A. Gingras
  4. B. Manning
  5. Guangchao Sui
  6. S. Hawley
  7. D. Hardie
  8. R. Weinberg
  9. D. Hanahan
  10. William C. Hahn

Who trusts D. Fingar overall

  1. A. Budanov
  2. K. Inoki
  3. Jingxiang Huang
  4. M. Karin
  5. Marina K. Holz
  6. B. Manning
  7. J. Blenis
  8. D. Munn
  9. N. Hay
  10. L. Bar-Peled

Papers from D. Fingar's perspective

Ordered by how strongly the researchers D. Fingar trusts endorse each paper. Same corpus, a different order for every researcher.

  1. TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
  2. Regulation of cellular growth by the Drosophila target of rapamycin dTOR. (Genes & Development)
  3. Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (Molecules and Cells)
  4. Enhanced sensitivity of PTEN-deficient tumors to inhibition of FRAP/mTOR (Proceedings of the National Academy of Sciences of the United States of America)
  5. Raptor, a binding partner of target of rapamycin (TOR), mediates TOR action. (Cell)
  6. The Mammalian Target of Rapamycin (mTOR) Partner, Raptor, Binds the mTOR Substrates p70 S6 Kinase and 4E-BP1 through Their TOR Signaling (TOS) Motif* (Journal of Biological Chemistry)
  7. Role of AMP-activated protein kinase in mechanism of metformin action. (Journal of Clinical Investigation)
  8. Mammalian TOR: A Homeostatic ATP Sensor (Science)
  9. A direct linkage between the phosphoinositide 3-kinase-AKT signaling pathway and the mammalian target of rapamycin in mitogen-stimulated and transformed cells. (Cancer Research)
  10. A DNA vector-based RNAi technology to suppress gene expression in mammalian cells (Proceedings of the National Academy of Sciences of the United States of America)

Their papers