D. Munn
Immunology · 2002–2016
A researcher in the Science corpus: 11 papers; trusts 25 researchers and is trusted by 13 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 126 citations into it; #398 of 2,160 by trust here, #523 by citations
- Molecular Biology — 51 citations into it
- Biological Psychiatry — 48 citations into it; #19 of 200 by trust here, #11 by citations
- Oncology — 36 citations into it
- Behavioral Neuroscience — 26 citations into it
- Genetics — 20 citations into it
- Psychiatry and Mental health — 17 citations into it
- Cell Biology — 15 citations into it
Whom D. Munn trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Francesca Fallarino
- H. Harding
- Madhav Sharma
- W. Zou
- A. Muller
- P. Rodriguez
- R. Wek
- Guido Kroemer
- A. Mellor
- Yuhong Zhang
Who trusts D. Munn overall
- Madhav Sharma
- A. Mellor
- W. Zou
- P. Rodriguez
- Erika L. Pearce
- B. Baban
- I. Mellman
- Francesca Fallarino
- Charles G. Drake
- D. Pardoll
Papers from D. Munn's perspective
Ordered by how strongly the researchers D. Munn trusts endorse each paper. Same corpus, a different order for every researcher.
- Evidence for a tumoral immune resistance mechanism based on tryptophan degradation by indoleamine 2,3-dioxygenase (Nature Medicine)
- Tolerance, DCs and tryptophan: much ado about IDO. (Trends in immunology)
- Expression of indoleamine 2,3-dioxygenase by plasmacytoid dendritic cells in tumor-draining lymph nodes. (Journal of Clinical Investigation)
- Ido expression by dendritic cells: tolerance and tryptophan catabolism (Nature reviews. Immunology)
- Regulatory T Cells Selectively Express Toll-like Receptors and Are Activated by Lipopolysaccharide (Journal of Experimental Medicine)
- Inhibition of Allogeneic T Cell Proliferation by Indoleamine 2,3-Dioxygenase–expressing Dendritic Cells (Journal of Experimental Medicine)
- Natural versus adaptive regulatory T cells. (Contributions to Nephrology)
- Regulated translation initiation controls stress-induced gene expression in mammalian cells. (Molecules and Cells)
- Perk is essential for translational regulation and cell survival during the unfolded protein response. (Molecules and Cells)
- T cell apoptosis by tryptophan catabolism (Cell Death and Differentiation)
Their papers
- Activation of the receptor (Gpr109a) for niacin and the commensal metabolite butyrate suppresses colonic inflammation and carcinogenesis (2014) — 2,096 citations
- IDO in the Tumor Microenvironment: Inflammation, Counter-regulation and Tolerance (2016) — 922 citations
- Indoleamine 2,3-dioxygenase is a critical resistance mechanism in antitumor T cell immunotherapy targeting CTLA-4 (2013) — 614 citations
- Indoleamine 2,3 dioxygenase and metabolic control of immune responses (2012) — 966 citations
- Coexpression of Tim-3 and PD-1 identifies a CD8+ T-cell exhaustion phenotype in mice with disseminated acute myelogenous leukemia. (2011) — 659 citations
- Plasmacytoid dendritic cells from mouse tumor-draining lymph nodes directly activate mature Tregs via indoleamine 2,3-dioxygenase. (2007) — 804 citations
- Indoleamine 2,3-dioxygenase and tumor-induced tolerance. (2007) — 1,038 citations
- GCN2 kinase in T cells mediates proliferative arrest and anergy induction in response to indoleamine 2,3-dioxygenase. (2005) — 1,238 citations
- Expression of indoleamine 2,3-dioxygenase by plasmacytoid dendritic cells in tumor-draining lymph nodes. (2004) — 908 citations
- Ido expression by dendritic cells: tolerance and tryptophan catabolism (2004) — 2,452 citations
- Potential Regulatory Function of Human Dendritic Cells Expressing Indoleamine 2,3-Dioxygenase (2002) — 1,089 citations