K. Taguchi

Molecular Biology · 2010–2012

A researcher in the Science corpus: 3 papers; trusts 3 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom K. Taguchi trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. K. I. Tong
  2. K. Itoh
  3. Masayuki Yamamoto
  4. N. Wakabayashi
  5. Donna D. Zhang
  6. A. Kobayashi
  7. Hozumi Motohashi
  8. T. Ohta
  9. M. Furukawa
  10. Moon-Il Kang

Who trusts K. Taguchi overall

  1. Q. Ma
  2. Feng He
  3. A. Dinkova-Kostova

Papers from K. Taguchi's perspective

Ordered by how strongly the researchers K. Taguchi trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
  2. Protection against electrophile and oxidant stress by induction of the phase 2 response: Fate of cysteines of the Keap1 sensor modified by inducers (Proceedings of the National Academy of Sciences of the United States of America)
  3. Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants (Proceedings of the National Academy of Sciences of the United States of America)
  4. BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
  5. Keap1 regulates both cytoplasmic‐nuclear shuttling and degradation of Nrf2 in response to electrophiles (Genes to Cells)
  6. Nrf2-Keap1 defines a physiologically important stress response mechanism. (Trends in Molecular Medicine)
  7. Molecular mechanism activating Nrf2-Keap1 pathway in regulation of adaptive response to electrophiles. (Free Radical Biology & Medicine)
  8. Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation (Nature Genetics)
  9. Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice. (Cell)
  10. Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)

Their papers