Q. Ma
Molecular Biology · 2013
A researcher in the Science corpus: 1 paper; trusts 27 researchers and is trusted by 6 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 46 citations into it; #3,722 of 17,773 by trust here, #11,818 by citations
- Epidemiology — 6 citations into it
- Physiology — 6 citations into it
- Cell Biology — 5 citations into it
- Immunology — 5 citations into it
Whom Q. Ma trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- T. Kensler
- Toren Finkel
- R. Scarpulla
- X. Wang
- Masayuki Yamamoto
- K. Taguchi
- A. Dinkova-Kostova
- J. Tschopp
- K. I. Tong
- Hozumi Motohashi
Who trusts Q. Ma overall
- Peter F Surai
- R. Kang
- Guido Kroemer
- Xiaofang Sun
- D. Tang
- M. Kerins
- Montserrat Rojo de la Vega
- J. Hayes
Papers from Q. Ma's perspective
Ordered by how strongly the researchers Q. Ma trusts endorse each paper. Same corpus, a different order for every researcher.
- Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
- Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation (Nature Genetics)
- Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)
- Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
- Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell Survival (Molecular and Cellular Biology)
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
- Keap1 Recruits Neh2 through Binding to ETGE and DLG Motifs: Characterization of the Two-Site Molecular Recognition Model (Molecular and Cellular Biology)
- p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription* (Journal of Biological Chemistry)
- Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis (Nature)
- Sensitivity to carcinogenesis is increased and chemoprotective efficacy of enzyme inducers is lost in nrf2 transcription factor-deficient mice (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Role of nrf2 in oxidative stress and toxicity. (2013) — 4,437 citations